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Management of eating disorders for people with higher weight: clinical practice guideline



The prevalence of eating disorders is high in people with higher weight. However, despite this, eating disorders experienced by people with higher weight have been consistently under-recognised and under-treated, and there is little to guide clinicians in the management of eating disorders in this population.


The aim of this guideline is to synthesise the current best practice approaches to the management of eating disorders in people with higher weight and make evidence-based clinical practice recommendations.


The National Eating Disorders Collaboration Steering Committee auspiced a Development Group for a Clinical Practice Guideline for the treatment of eating disorders for people with higher weight. The Development Group followed the ‘Guidelines for Guidelines’ process outlined by the National Health and Medical Research Council and aim to meet their Standards to be: 1. relevant and useful for decision making; 2. transparent; 3. overseen by a guideline development group; 4. identifying and managing conflicts of interest; 5. focused on health and related outcomes; 6. evidence informed; 7. making actionable recommendations; 8. up-to-date; and, 9. accessible. The development group included people with clinical and/or academic expertise and/or lived experience. The guideline has undergone extensive peer review and consultation over an 18-month period involving reviews by key stakeholders, including experts and organisations with clinical academic and/or lived experience.


Twenty-one clinical recommendations are made and graded according to the National Health and Medical Research Council evidence levels. Strong recommendations were supported for psychological treatment as a first-line treatment approach adults (with bulimia nervosa or binge-eating disorder), adolescents and children. Clinical considerations such as weight stigma, interprofessional collaborative practice and cultural considerations are also discussed.


This guideline will fill an important gap in the need to better understand and care for people experiencing eating disorders who also have higher weight. This guideline acknowledges deficits in knowledge and consequently the reliance on consensus and lower levels of evidence for many recommendations, and the need for research particularly evaluating weight-neutral and other more recent approaches in this field.

Plain English summary

The objective of this project was to develop recommendations and clinical considerations to guide clinicians in the management of people experiencing eating disorders who also have higher weight. A Guideline Development Group was formed containing members with academic and/or clinical expertise and people with a lived experience of eating disorder. The guideline was not only informed by reviews of the scientific literature but also clinical expertise and lived expertise. This guideline has undergone extensive review and consultation over an 18-month period involving reviews by key stakeholders, including experts and organisations with clinical, academic and/or lived expertise. The guideline outlines a set of recommendations for clinical practice including the strong recommendation for psychological treatment to be offered as the first treatment for an eating disorder in people who are of higher weight. Considerations in clinical practice including weight stigma, care by professionals from disparate disciplines, and cultural considerations are also discussed. The Guideline Development Group acknowledges a lack of available research evidence specific to people experiencing an eating disorder who are also of higher weight and consequently some recommendations relied on consensus of group members taking into account the expert reviews. The Group also identified areas where additional research is necessary such as research evaluating weigh-neutral and other more recent approached in the field.


Executive summary

Eating disorders are serious, complex and potentially life-threatening mental illnesses. While historically, eating disorders have been conceptualised as disorders of people of low body weightFootnote 1 there is now substantive evidence that this is inaccurate. The most common eating disorders are binge-eating disorder, other specified feeding or eating disorder (OSFED) and bulimia nervosa, and these occur in people across a broad range of body types [1]. Eating disorders are common and increasing in prevalence. This is particularly true for people with eating disorders who are of higher weight. This population comprises more than half of all people with an eating disorder in Australia with rates of eating disorders increasing most in people with higher weight [2].

A key rationale for this guideline (see Box 1) is that despite the high prevalence, eating disorders in people with higher weight have been consistently under-recognised and under-treated. People with a lived experience of an eating disorder who are of higher weight report being misdiagnosed, dismissed by health professionals and sidelined or excluded from eating disorder treatment services. This population is also often absent from eating disorders research, with the exception of binge-eating disorder. Weight stigma is a major factor contributing to these shortfalls and is addressed in this guideline. This guideline aims to promote weight-inclusive practice and provide advice on how to avoid weight stigmatising practices for people with an eating disorder who are of higher weight.

Box 1 Lived experience perspectives: why we need this guideline

The aim of this guideline is to synthesise the current best practice approaches to the management of eating disorders for people who are of higher weight, based on the premise that every person with an eating disorder is deserving of equitable, safe, accessible, and evidence-based care regardless of their body size. It accords with the role and function of the National Eating Disorders Collaboration (NEDC) to synthesise research evidence, clinical expertise and lived experience in national standards to improve systems of care for all Australians. While it is important to recognise eating disorders in people with ‘severe obesity’ or those presenting for bariatric surgery, it is important to note that the aim of this guideline is not to address weight loss or treatment of ‘obesity’.

In 2019, the NEDC Steering Committee auspiced this guideline and a Guideline Development Group was formed containing academic and/or clinical expertise, and/or lived experience from diverse disciplines. Modelled on the ‘Guidelines for Guidelines’ process outline by the National Health and Medical Research Council (NHMRC), the guideline was not only informed by recent systematic reviews, meta-analyses and primary trials, but also clinical expertise and lived expertise. It should also be noted that the voice of lived experience is largely absent in the literature. This guideline has undergone extensive peer review and consultation over an 18-month period involving reviews by key stakeholders, including experts and organisations with clinical and/or academic expertise and/or lived experience.

This guideline is intended for all health care professionals and does not present specialist information for any specific discipline. It does not aim to provide recommendations on prevention or detection but does provide advice on assessment. The guideline addresses treatment and/or management recommendations, specifically for people with an eating disorder who are of higher weight. This encompasses, but is not limited to psychological, pharmacological, nutritional, medical, family and activity interventions. Management should address all aspects of an eating disorder, thus interprofessional collaborative practice (ICP) is recommended, with each clinician practicing within the scope of their profession. Readers are referred to other literature for management of specific medical and other psychological disorders that are often experienced by people who have an eating disorder and are of higher weight.

It is hoped that this guideline will assist health care professionals in all relevant fields to understand the needs of people in their care who have an eating disorder who are of higher weight, and support the clinician in providing appropriate management of the eating disorder. Moreover, it is hoped that clinicians are more aware of, and responsive to, the adverse effects of weight stigma on the lives, health and treatment seeking of people with eating disorders who are of higher weight. This guideline acknowledge deficits in knowledge and consequently the reliance on consensus and lower levels of evidence for many recommendations, and the need for research particularly evaluating weight-neutral and other more recent approaches in this field.


The aim of this guideline is to synthesise the current best practice approaches to management for people with an eating disorder who are at higher weight. The focus is on the treatment of the eating disorder (see “Background to eating disorders and how they occur” section for a definition of eating disorders), experienced in people living with higher body weight. The aim is not to address weight loss or “treatment of obesity”.

This guideline is intended for all health care professionals and does not present specialist information for any specific discipline. Where applicable, readers will be directed to resources for the latter. It is also not aiming to provide formal recommendations on prevention but does discuss clinical considerations of identification and assessment.

This guideline was developed within the Australian context and thus includes reference to Aboriginal and Torres Strait Islander peoples. However, it is anticipated to be relevant more widely as representing current knowledge and best health practice broadly. For this reason we have chosen to publish in international literature where it comes under scrutiny with international review. As the focus of this guideline is on the management of eating disorders, the outcomes considered are those relevant to the eating disorder. General physical and mental health-related quality of life are relevant as secondary outcomes. A reduction in body weight, or stabilisation of fluctuating body weight in itself is not an outcome or goal of treatment of an eating disorder experienced by people with higher weight. Further, it is possible that attempts at weight loss may exacerbate eating pathology and therefore may be contraindicated in some people (see Box 2).

Box 2 Weight loss and health in people with higher weight

Notwithstanding that, we acknowledge that some people with significant medical co-occurring conditions or those presenting for bariatric surgery may require and seek significant weight loss, in the presence of diagnosed or undiagnosed eating disorders. We also acknowledge the complexities for people experiencing an eating disorder who are undergoing bariatric surgery and other weight loss regimes. While the management of obesity is not within the scope of this guideline, it is hoped that this guideline will assist health care professionals in all relevant fields to understand the needs of people in their care with an eating disorder, refer appropriately, and work collaboratively with other health professionals providing care and treatment for people experiencing eating disorders.

Weight stigma

Weight stigma is the disparaging association of higher weight with negative personal characteristics [3]. ‘Weight stigma’ in this guideline is used to mean the occurrence of discrimination against or stereotyping of a person based on their weight, size or shape [4]. Other terms used are ‘sizeism’, ‘weight/size oppression’, ‘weightism’, ‘weight/size bias’, ‘weight-based discrimination’ and ‘fat phobia’.

Internalised weight stigma occurs when an individual upholds these disparaging associations towards their own body weight. Stronger internalised weight stigma predicts greater eating disorder psychopathology, higher levels of body dissatisfaction and poorer quality of life [5] and is common among people seeking bariatric surgery [6]. Stigma may also extend to the negative impacts of weight-stigma in parents of higher weight children [7].

Weight stigma has serious adverse impacts on the lives, health and treatment seeking of people with higher weight. Weight stigma may lead directly to disordered eating via complex neurobiological mechanisms, or with the aim of reducing the emotional distress it causes [8, 9]. There is active investigation into neurobiological mechanisms of weight stigma and the relationship with disordered eating [e.g., the research of 911]. Understanding and addressing weight stigma is crucial to the care of people with higher weight. Experiences of weight stigma, body shame or other negative emotions such as guilt are traumatic and may contribute to the onset of eating disorders and increase disordered eating in those with eating disorders [12,13,14]. Perceiving and experiencing a health care provider as weight stigmatising is associated with disengagement from treatment or health care [15, 16].

An important aspect in addressing weight stigma is in the use of language that avoids stigmatising terms for someone experiencing weight stigma. For this reason, this guideline use the phrases ‘people with higher weight’ and ‘living in a larger body’. Notwithstanding this approach, it is important to emphasise that there is not one universally preferred term for people living in larger bodies and health professionals should discuss preferred language with each person.

Despite being recognised for nearly half a century [17] weight stigma continues to be a major factor in the under-recognition and under-treatment of eating disorders, and especially of eating disorders experienced by people with higher weight. It is not well understood by the broader medical community that eating disorders among people of higher weight are just as serious and life threatening (from medical complications and self-harm) as eating disorders among people at lower weight. In addition, eating disorders at any weight are associated with a high level of psychological distress and psychopathology [18,19,20]. In reviewing the literature for this guideline, it is notable that the bias applies in both directions. That is, there are major gaps in the literature pertaining to both the treatment of binge-eating disorder (BED) for people at any size, and, more relevant to this guideline, the treatment of eating disorders other than BED in people at higher body mass indexes (BMIs).

Health professionals may be influenced by societal views on higher body mass and offer treatment tailored to a person’s weight rather than their eating disorder (e.g., advising a medication for its appetite suppressing effects rather than binge eating reduction). Health professionals need to be aware of the risks versus benefits of discussing body weight, particularly with people vulnerable to, or who have experienced an eating disorder. This guideline aims to promote weight-inclusive practice and advice on how to avoid weight stigmatising practices for people with an eating disorder who are of higher weight.

Limitations of body mass index (BMI),Footnote 2 language and definition of key terms

Cognisant of weight stigma and other considerations in this guideline, the terms larger bodied and higher weight includes people with high body mass index (BMI; kg/m2) through low adiposity and high muscle density (i.e., muscle building/athletes in larger bodies), as well as those with high adiposity. It may also include people with high adiposity but normal metabolic health indices and no physical heath co-occurring conditions [27] although these may develop in the future. Thus, this guideline does not define higher weight by a BMI cut off but rather focusses on a conceptualisation of a larger body that includes people who may be impacted socially and by the health system by standard BMI cut off points.

Historically BMI has been and continues to be widely used as an indicator of risk relating to physical health status. However, it is acknowledged that there are limitations to sole reliance on BMI [28]. As noted above, body composition can be highly variable in people with the same BMI and is influenced by many factors such as age, sex, race and muscularity. BMI has utility as a chronic disease risk marker in a population but should be used with other indicators of health status for a person. In individual assessment, other anthropometric, biochemical and behavioural measures may include waist circumference, blood pressure, blood glucose and lipid profiles. In children and adolescents, the height and weight growth velocity is preferred to the BMI. For all people it is more useful, if possible, to consider the person’s pre-illness growth trajectory as likely to be close to their ‘normal’ or ‘natural’ body habitus. This trajectory should be used to guide assessments of nutritional repletion and physical recovery. It is also important to note that people living in larger bodies, may have been engaged in weight suppression strategies for many years (in some instances, since childhood), and prior to the eating disorder, and thus their pre-illness BMI may yet be weight-suppressed rather than ‘natural’.


Rationale for this guideline

Historically, eating disorders have been conceptualised as illnesses of people of low body weight [1] and typified by disorders such as anorexia nervosa. There is now substantive evidence that this is inaccurate. The most common eating disorders are BED, other specified feeding or eating disorder (OSFED)Footnote 3 and bulimia nervosa, and these occur in people across a broad socio-demographic spectrum and a range of body types. This guideline address the particular issues that arise in the care of people experiencing eating disorders who are of higher weight. These individuals represent over half of all people experiencing an eating disorder in Australia with rates of eating disorders increasing most in people with higher weight [2]. The issues affecting people with eating disorders who are of higher weight are complex and important. These issues include delayed identification, misdiagnoses in assessment, subsequent inappropriate and inadequate treatment, widespread stigma, and the introduction of new disorders (i.e., anorexia nervosa without low weight). To our knowledge there are no current Australian guidelines to assist health professionals caring for people with both eating disorders and higher weight.

Background to eating disorders and how they occur

The main DSM-5 eating disordersFootnote 4 are described in Table 1. They comprise anorexia nervosa, bulimia nervosa, BED, avoidant restrictive intake disorder (ARFID), OSFED and unspecified feeding or eating disorder (UFED). Only one, anorexia nervosa, is defined by weight (i.e., underweight criteria). Where all features of anorexia nervosa are present except for low body weight, DSM-5 suggests a diagnosis of ‘atypical anorexia nervosa’. In most respects the World Health Organization ICD-11 [29] criteria closely match those of the DSM-5, though the ICD-11 does not require low-weight for a diagnosis of anorexia nervosa. For the purposes of this guideline, when providing advice on assessment or recommendations for treatment, the ICD-11 terminology for anorexia nervosa is adopted. That is, anorexia nervosa (code 6B80) is used as a broad term to include people at all body weights and without specifying the underweight criterion (sub coded in ICD-11 as 6B80.0, anorexia nervosa with significantly low body weight). The other eating disorders can occur in individuals across the weight spectrum.

Table 1 Overview of DSM-5 diagnostic criteria for eating disorders

Eating disorders are common and increasing in prevalence. There is a lifetime estimated prevalence of 8.4% for women and 2.2% for men [30]. In Australia, the 3-month point prevalence is around 0.5% for low weight anorexia nervosa, 1% for bulimia nervosa and 1.5% for BED (broadly defined with ICD-criteria) and 3.2% for OSFED [including anorexia nervosa (without low weight) prevalence of 2.5%]. Furthermore, around 10% of people have recurrent binge eating [31] with rates of binge eating increasing most in people with higher weight [2]. A recent meta-analysis suggested lower rates of eating disorders but this may be accounted for by 25% of included studies being from China with large samples and generally low identification of eating disorders in these studies other than anorexia nervosa [32].

Eating disorders are also prevalent in diverse populations including men [33], across sexual and gender minority identities [34], all levels of socioeconomic status [35] and, migrant status [36]. Whilst more prevalent among adolescents and young people, they can affect people at any age including middle-aged and older adults [35, 37]. There is limited research on the experience of eating disorders in Aboriginal and Torres Strait Islander peoples. However, emerging research suggests that eating disorders are more common in Aboriginal and Torres Strait Islander adults and youth compared with non-Indigenous people [38].

Eating disorders have complex biological, social, and psychological determinants [39]. These include strong heritability and a range of risk factors that are common to and overlap with a predisposition to a higher body size, such as a personal history of trauma (see Box 3) in the formative years of life [40,41,42]. For people with higher weight, recommendations for weight loss by health professionals without sufficient monitoring, may be associated with the onset of an eating disorder, especially in adolescents [43].

Box 3 Trauma-informed care

Eating disorders have severe psychological, medical, community, public health, and fiscal consequences [44] with the highest mortality rates of any mental disorder [45] and high global burden—an estimated 6.6 million disability-adjusted life years [46]. Psychological comorbidity occurs in over 80% of people with eating disorders, and more specifically, in over 90% of people with bulimia nervosa or BED. Over 50% of people with bulimia nervosa or BED may have a major depressive disorder, followed by persistent depression, and around 40–50% have experienced anxiety disorders (most commonly generalised anxiety disorder). Also occurring frequently are posttraumatic stress disorder, substance use disorder (particularly alcohol use disorder), followed by a personality disorder [47]. Physical co-occurring conditions are also common. In the Udo and Grilo (2019) study [47], disorders associated with the metabolic syndrome such as hyperlipidaemia and diabetes mellitus were particularly common, as well as musculoskeletal disorders such as arthritis, fibromyalgia, and sleep problems in people with binge-eating disorder. Osteoporosis was most prevalent in people with low weight anorexia nervosa but also occurred in 6.1% of people with BED, where bowel problems (e.g., inflammatory bowel disease and irritable bowel syndrome) were also higher (around 11%) than in people without an eating disorder.

Current status of treatment and outcomes for all eating disordersFootnote 5

Psychological: first line

The first line outpatient treatment for any person with an eating disorder is an evidence-based psychological therapy delivered by an eating disorder informed and trained therapist [48, 49]. The therapies are described in Table 2. Whilst there are distinct features of these therapies, it should be noted that there are many common elements including but not limited to addressing body image (see Box 4).

Table 2 Overview of main psychological therapies for the management of low weight anorexia nervosa, bulimia nervosa and binge-eating disorder
Box 4 Body image

Psychological therapies in adults include: cognitive behaviour therapy-enhanced (CBT-E); cognitive behaviour therapy for anorexia nervosa (CBT-AN); Maudsley model of anorexia nervosa treatment for adults (MANTRA), specialist supportive clinical management (SSCM); focal psychodynamic therapy (FPT); interpersonal psychotherapy (IPT); family based treatment; and dialectical behaviour therapy (DBT). Only one, CBT-E is ‘transdiagnostic’ (i.e., has an evidence-base for use in adults with anorexia nervosa, bulimia nervosa, BED and OSFED types). They are all manualised. Some have been evaluated in group, internet and self-help formats. In particular, cognitive behaviour therapy (CBT) for BED and bulimia nervosa may be delivered by primary care therapists in a guided self-help form. However, abstinence and attrition rates are superior in traditional psychological therapy and guided self-help versus pure self-help modes [49].

Children and youth

Family involvement in the treatment of children and adolescents at all levels of care is developmentally appropriate and best practice. A special form of family therapy with a specific eating disorder focus first developed in the UK and later the US (often referred to as the Maudsley model, family based treatment or family therapy for anorexia nervosa) is first line for children and adolescents with low weight anorexia nervosa and has been adapted for use in other eating disorders such as bulimia nervosa [49,50,51,52]. Family therapy (FBT/FT-AN) aims to establish parental management of their child’s nutritional recovery before focussing on other psychological and psychosocial issues. It has been found to be effective in a number of randomised controlled trials (RCTs) and is supported by a recent systematic review [53]. If family therapy (FBT/FT-AN) is contraindicated owing to family availability or safety concerns, then a second line treatment should be considered. High levels of family involvement in inpatient and day patient settings are usually a standard part of any program [c.f. 5456]. Recent research has also explored the use of FBT for transition age youth (17–25 years) with anorexia nervosa, but with a more collaborative stance between parents and the young person that reflects their age [57]. The evidence-base for FBT in this age group is yet to be established.

While there is less evidence for the treatment of adolescents with bulimia nervosa in comparison with low weight anorexia nervosa, the current first line treatment for adolescents with bulimia nervosa is also FBT [58]. Family interventions for BED are yet to be studied. Alongside the published manuals for anorexia nervosa and bulimia nervosa there is also an FBT manual specific to ARFID [59] and a manualised form of CBT developed for children and adolescents with ARFID (CBT-AR) that can be delivered in individual or family based formats [60]. It is undergoing evaluation. People with OSFED are usually treated with the therapy corresponding to the full syndrome (e.g., subthreshold bulimia nervosa and bulimia nervosa).

Family specific interventions for BED are yet to be studied but there are some promising applications of IPT [61] emerging in the literature that focus on preadolescents vulnerable to developing excessive weight gain and BED. In both individual and family formats, IPT has led to improvement in internalising symptoms thought to lead to a loss of control, a symptom of BED. These are promising results given the importance of early intervention in the development of an eating disorder. In addition to IPT there is emerging evidence for CBT and DBT for BED in adolescents. CBT has been shown to be effective when compared to a weight loss treatment at both end of treatment and in the longer term [62] and DBT in reducing BED symptoms, although was not more effective than behavioural weight loss [63].

While family based treatments remain first line for anorexia nervosa and bulimia nervosa there is a need for other treatments to emerge that can specifically address other eating disorders (i.e., ARFID, BED) and non-responders in a similar evidence based way. Current recommended second line treatments for children and adolescents are noted in the next section.

Psychological and other: second line treatments


For adults who have difficulty accessing a first line therapy and/or who do not respond, or only have partial improvement, a second line treatment may be considered. Second line psychotherapies in adults include ‘third-wave’ [64] psychological therapies such as mindfulness-based therapy and Acceptance and Commitment Therapy (ACT). These have less evidence of efficacy compared to first line treatments, but may be helpful options when first line treatments have not been effective.

A psychological therapy informed by weight neutral practice and Health at Every Size® (HAES) principals (J.L Gaudiani, personal communication to author PH, August 21, 2021) has been developed with one open unpublished report (see “Psychological therapy for adults” section later in this document). It is based on an understanding that body dissatisfaction emerges in the context of weight stigma, and both are important predisposing, precipitating and perpetuating factors in eating disorders; it thus comprises weightinclusive and trauma-informed care where body acceptance (amongst others) is a protective factor.

Family interventions for adults with an eating disorder are less common and none are currently recommended as first line treatment [49]. However, some family inclusive interventions have been evaluated. The most established is Maudsley collaborative care [65, 66]. This model educates carers of adults with anorexia nervosa to support their loved one with strategies that target maintaining aspects of the illness. Parts of this intervention are also part of MANTRA, a firstline therapy (see Table 2). Other such approaches include the addition of family therapy or couple therapy alongside individual therapy [67,68,69] as well as group-based programs for carers.

Multiple family therapy has also been shown to be feasible with adults with anorexia nervosa [57, 70, 71]. Most studies to date report the inclusion of families in the treatment of adults with anorexia nervosa, but a recent study by Runfola et al. [72] tested a model for couple therapy specifically designed for BED in a small open trial and was found to be feasible.

Second line psychotropic medications include antidepressants, antipsychotics, psychostimulants and anticonvulsants. Their main use is summarised in Table 3. All psychotropic medications have potential to impact on appetite and body weight (though our current understanding of these effects is poor). They are seldom considered as a stand-alone treatment in eating disorders particularly because risk of relapse when discontinued and are most often prescribed as adjunctive to psychological therapy [48].

Table 3 Psychotropic medications commonly used in anorexia nervosa, bulimia nervosa and binge-eating disorder
Children and youth

In children and adolescents, where family therapy is not available or inappropriate, the two most common second line treatments for anorexia nervosa [49] are CBT-E for adolescents [73, 74] and adolescent focused therapy [AFT; 75]. Parent and family sessions should be offered alongside the individual sessions. Other commonly utilised interventions involving families for children and adolescents include multifamily group programs [76] and parental psychoeducation [77] as adjunctive to a first line intervention.

Other treatments

Behavioural weight loss intervention (BWLI) is a comprehensive psychobehavioural treatment with activity and nutrition therapy developed for people with higher weight that has since been tested as an active and as a control psychological therapy for people with recurrent binge eating and other eating disorders and found to be efficacious. In the short-term, binge eating frequency improves but in the longer term, maintenance of change is less clear [78].

Exercise and its management in general eating disorder populations (largely focusing on bulimia nervosa and low weight anorexia nervosa) is mainly targeted at reducing compulsive overexercise [79]. These interventions typically include structured physical activity under supervision (often in a group setting) and individual psychotherapy, and demonstrate improvements in depressive symptoms, skeletal muscle mass and quality of life [80, 81]. Interestingly, effects on exercise compulsion have been mixed [82]. Dittmer et al. [83] found a significant reduction in compulsive exercise in their intervention for inpatients with low weight anorexia nervosa, whilst Mathisen et al. [84] and Zeeck et al. [82], found no significant reductions compared with control groups. In contrast, Ng et al. [85] and Moola et al. [86] found that compared to a control group, people with low weight anorexia nervosa undertaking prescribed exercise reduced eating disorder symptoms, including disordered beliefs about food and exercise, and enhanced quality of life.

More recently, there have been some RCTs of neuromodulation treatments for people with eating disorders such as low weight anorexia nervosa, bulimia nervosa and BED. Treatments such as repetitive transcranial magnetic stimulation (rTMS) may aid in reducing symptoms such as binge eating and improving appetite regulation and mood [87]. As of writing this guideline they remain experimental treatments for eating disorders in Australia.

Psychological co-occurring conditions

Notwithstanding the need for evidence-based eating disorder treatment many people may also require psychological or other treatments for common co-occurring conditions such as major depression, anxiety disorders and/or substance-use disorder. Psychological therapy for people with eating disorders may also benefit from a trauma-informed care (see Box 3) or specific therapy such as eye movement desensitisation and reprocessing (EMDR) for post-traumatic stress disorder [88].

Physical co-occurring conditions and consequences

Physical co-occurring conditions in people experiencing an eating disorder, with or without a high body weight, are common. In a national US sample of 36,309 adults (NESARC-III),Footnote 6 more than half of those with an eating disorder reported at least one chronic medical condition diagnosed within the previous 12 months (54.5 ± 5.1% for bulimia nervosa and 68.6 ± 63.0 for BED), as seen in Tables 3 and 4 of Udo and Grilo [47; see further 103, 104]. Prevalence of co-occurring somatic conditions is outlined in Box 5.

Table 4 Assessment instruments recommended for use with people with higher weight
Box 5 Prevalence of co-occurring somatic conditions across DSM-5 bulimia nervosa and binge-eating disorder across the BMI spectrum

While higher weight has been linked to various co-occurring somatic conditions, a review by Olguin et al. [105], discussed cross-sectional epidemiologic data that showed BED was associated with diabetes, hypertension, dyslipidaemias, sleep problems/disorders, and pain conditions, and that BED may be related to these conditions independent of BMI or co-occurring psychiatric disorders. Prospective data suggest that BED may be associated with type 2 diabetes and metabolic syndrome independent of weight. BED and binge eating behaviour are also associated with asthma and gastrointestinal symptoms and disorders, and among women, menstrual disruption, pregnancy complications, intracranial hypertension, and polycystic ovary syndrome (PCOS).

The consequences of bulimia nervosa are similar regardless of BMI. These consequences include the physical effects of purging, which can affect the skin, teeth, eyes/ears and nose, throat, gastrointestinal tract, electrolytes, heart, a possible increase risk of miscarriages, and a rare risk of aspiration pneumonia [106].

People with eating disorders who restrict their dietary intake and/or engage in other behaviours such as purging may experience malnutrition resulting from poor dietary quality leading to altered body composition and body cell mass, and diminished physical and mental function and impaired clinical outcomes [107, 108] Further, the severity of the eating disorder in anorexia nervosa (without low weight) is more closely related to the amount and rapidity of weight loss and weight suppression (which may be seen also in BED and bulimia nervosa) than the actual admission weight or BMI in adolescents and physical consequences may be similar to low weight anorexia nervosa [19, 109].

Acute medical issues and admission

People with an eating disorder at any weight may need admission to a medical or psychiatric ward to stabilise very severe eating disorder symptoms (e.g., very frequent binge eating) and/or to reverse a starvation state or acute medical complications such as low potassium levels [see RANZCP guidelines; 112]. People may also have a co morbid medical or psychological complication requiring acute care (e.g., unstable diabetes or suicidal ideation with intent).

Methods of guideline development

Aim and method


The aim of this guideline is to synthesise the current best practice approaches to the management of eating disorders for people who are of higher weight. The focus is on the treatment of the eating disorder, with consideration of higher weight. The aim is not to address weight loss or treatment of obesity. The guideline provides guidance on providing treatment for people currently with higher weight whether or not the eating disorder developed when the person was of a higher weight.

Formation of the guideline development group

The National Eating Disorders Collaboration (NEDC) synthesises research evidence, clinical expertise and lived experience in national standards and workforce initiatives to build and effective, equitable and accessible system of care for all Australians. This guideline received funding from the Australian Government Department of Health. The NEDC Steering Committee agreed to auspice this guideline in 2019 and members of the Steering Committee with diverse discipline specific expertise volunteered to comprise a Writing Group. Members of the Writing Group included individuals with lived experience and/or clinical expertise and/or research expertise. At the first meetings of the Writing Group, additional members were invited into the Writing Group so representatives were included to reflect disciplines and expertise not already within the group. A wider group was then formed, namely the Guideline Development Group. This comprised the members of the Writing Group as well as additional people with lived experience who had diverse demographic characteristics (e.g., gender; Aboriginal and Torres Strait Islander status) as well as varied experiences of eating disorders, such as different diagnoses and roles (i.e., whether they had a personal lived experience of an eating disorder or were a family member or support for someone with an eating disorder). Membership was approved by the NEDC Steering Committee and NEDC National Director.

Guideline Development Group Members’ curriculum vitaes are found in Additional file 1: Appendix A along with members’ declarations of interest at the end of this document.

Process of guideline development

The Guideline Development Group followed the process outlined in Box 6 which is modelled on the ‘Guidelines for Guidelines’ process outlined by the National Health and Medical Research Council [NHMRC; 113]. The Group also followed the RIGHT (Reporting Items for Practice Guidelines in Healthcare) Statement for Practice Guidelines [Additional file 2: 114 ]. Decisions were made by consensus in consideration of identified evidence, and expertise and experience of members.

Box 6 Process of guideline development

NEDC intends to update this guideline in 2025.

Research evidence

The guideline was informed by recent systematic reviews and meta-analyses as well as identified primary trials. With regard to psychological interventions for eating disorders in people with a higher weight, evidence was specifically sourced from the results of a systematic review and meta-analyses [115; manuscript in preparation]. Systematic reviews and meta-analyses were identified through a systematic literature search, existing guidelines, personal libraries of authors and additional papers identified by expert reviewers. The quality of systematic reviews and meta-analyses was critically appraised using the JBI critical appraisal checklist for systematic reviews and research syntheses [116]. The appraisal was conducted independently by author AR and contributor KP and disagreements were resolved by consensus (Additional file 3).

A full list of all the meta-analyses, systematic reviews and identified primary trials not included in a referenced systematic review used to inform this guideline is provided in Additional file 1: Appendix B. Recommendations were graded according to NHMRC categories A–D (Additional file 1: Appendix C).

It should be noted that there is a paucity of research that includes the voice of people with a lived experience.

Lived experience contribution

In addition to the lived experience representatives within the Guideline Development Group, further lived experience expertise was sought to co-write sections of this guideline for specific considerations for LGBTIQA+ people and Aboriginal and Torres Strait Islander peoples (see “Cultural considerations” section). We acknowledge that there is great diversity of all peoples’ lived experience, in particular, within Aboriginal and Torres Strait Islander peoples, exemplified by over 250 different languages across Australia. Moreover, we acknowledge intersectionality of people’s experiences and identities, that is, that people may belong to more than one minority group and that this may compound the difficulties they experience. Thus the views represented within this document may not capture this diversity.

Culturally informed practice

At the time of writing this guideline, it was apparent that there are significant gaps in the understanding and development of culturally informed assessment and treatments for larger-bodied Aboriginal and Torres Strait Islander peoples with eating disorders. When working in Australia, health professionals at all levels of experience should have received training in culturally informed practice particularly when working with Aboriginal and Torres Strait Islander peoples. This is also important to consider when working with people from culturally and linguistically diverse backgrounds and other minority groups (such as LGBTQAI+ people).


Identification and assessment

People of higher weight are at increased risk of eating disorders compared to those with lower weight [117], but due to many reasons, including poor health literacy (e.g., lack of understanding that eating disorders occur across the weight spectrum) and weight stigma in the community and in health care providers, their symptoms often go undetected and untreated (see Box 7 for a lived experience perspective) [118]. Early intervention provides the best chance of recovery when an individual is experiencing an eating disorder. Notwithstanding this, it is noted that approaches to screening have a very limited evidence-base particularly in children and adolescents, and more research is needed to establish risks and benefits [119, 120]. It is therefore imperative that eating disorder symptoms are identified and that intervention is offered as soon as possible [121] to all individuals experiencing eating disorder symptoms regardless of weight status.

Box 7 A lived experience perspective: identification and assessment

It is important to note that binge eating, loss of control, grazing or emotional eating are not the only or even predominant eating behaviours experienced among people with higher weight [2, 122]. Dietary restriction and other disordered behaviours (e.g., use of laxatives, purging, driven or compulsive exercise, dietary supplements use or abuse) are also frequently present among people with higher weight [2]. Notably, people with higher weight experience the cognitive factors associated with an eating disorder, including overvaluation of and preoccupation with weight, shape, eating and their control, and the distress associated with these cognitions. Warning signs and clinical considerations for eating disorders among people with higher weight are outlined in Box 8.

Box 8 Warning signs and clinical considerations for eating disorders among people of higher weight

When people living in larger bodies seek primary or mental health care for weight loss, assessment of eating disorder symptoms should be made. All services recommending or providing weight loss advice or programs (including bariatric surgery) should screen for disordered eating, risky behaviours such as use of unregulated weight-loss pills/supplements or laxatives, and body image concerns. All positive screens should be discussed with the individual and a more extensive eating disorders assessment should be undertaken. Health professionals in any setting should monitor any attempts at weight loss or muscle building. Short screening tools such as the Eating Disorder Screen for Primary Care [ESP; 123; see Additional file 1: Appendix D] may be also useful for this purpose. The components of a mental health assessment for eating disorders is detailed in Box 9.

Box 9 Mental health assessment for eating disorders

It is important to note that there is currently a lack of data regarding identification and assessment for underrepresented groups including males, adolescents, LGBTIQA+ people and people from cultural minority groups [119, 120]

Although body weight fluctuations can be a sign of an eating disorder, clinicians should not wait for body weight changes to occur before considering an eating disorder assessment.

Assessment of eating pathology in people with higher weight

Assessment of a person suspected to have an eating disorder should proceed in accordance with the Australia and New Zealand Academy for Eating Disorders Practice Standards 2020 [136]. Described here are particular considerations for the assessment of eating disorders among people with higher weight.

Because of wide-spread weight stigma in the community, people living in larger bodies often experience stigmatisation and discrimination because of their weight (i.e., weight teasing or bullying, negative interactions with family, friends, partners, co-workers, education or healthcare providers). Body dissatisfaction may be a natural consequence of ongoing negative evaluation rather than an irrational fear or distortion. People with higher weight also have often experienced weight-related trauma, such as bullying in high school or weight-related emotional abuse. Experiences of stigma and discrimination may lead to individuals being reluctant to talk about their weight or eating, for fear of being further shamed and/or disbelieved, and these issues must be approached respectfully, with consideration of prior negative experiences.

Disordered eating behaviours may function as a coping mechanism in the face of the trauma of persistent weight stigma. Severe dietary restraint and unhealthy compensatory behaviours may have been positively rewarded and reinforced by an individual’s social network or health professionals The person may therefore be reluctant to disclose compensatory behaviours, over-eating, or to make changes to any weight loss strategies, even though these strategies may be harmful. As opposed to attitudes of concern expressed towards smaller-bodied people engaging in dietary restriction, larger-bodied people engaging in the same or more severe degrees of restriction are often commended and encouraged to continue, with many eating disorder symptoms being perceived as helpful to the achievement of a weight loss goal. Further, where qualifying for surgery or other interventions requires the absence of eating disorder symptoms, clinicians must be cautious in their assessment of a person’s presentation.

For all people with an eating disorder, information on eating, purging and compensatory behaviours may need to be gathered from multiple sources, including family and supports, especially among children and adolescents. Eating psychopathology can impair perceptions of frequency of disordered behaviours or amount of food intake, so verification with other sources can be useful for establishing clinical status. However, for people with higher weight, it is important not to assume that the person is being untruthful. Instead, be respectful and sensitive when gathering information, even with the knowledge that a person may minimise their symptoms for fear of losing important coping mechanisms or access to interventions. The way clinicians approach questioning about eating habits and compensatory behaviours is critical to establishing a non-stigmatising and supportive therapeutic alliance. This includes respectfully seeking permission to obtain further information from family or others.

It is important not to make assumptions about a person’s eating or compensatory behaviours on the basis of weight. For example, do not assume that the person is engaging in binge eating, is untruthful about their dietary intake, or is not restricting. There are a wide range of eating disorder presentations among those living in larger bodies, including severe dietary restriction, and all possible diagnoses should be assessed before being ruled out.

A comprehensive assessment of the individual and their circumstances should be undertaken to confirm an eating disorder diagnosis and any co-occurring psychiatric or medical diagnoses, to evaluate medical and psychiatric risks, and to develop a biopsychosocial formulation. Collecting assessment information is an ongoing task as clinical issues and priorities unfold throughout treatment.

In some people with eating disorders, weight loss treatment may be contraindicated or may exacerbate their eating disorder. Where possible, attempts at weight loss or plans for bariatric surgery should be conducted in a setting to allow their eating disorder to be managed. Communication of diagnosis, medical and psychiatric risk, to other relevant treating professionals is therefore essential, especially where there are prescriptions for weight-loss treatments and/or plans for bariatric surgery. Referrals to support organisations for loved ones, family and parents are also recommended.

Assessment instruments

The ANZAED practice standards [137] recommend use of a psychometric assessment tool suitable for the assessment of eating disorders (using the Eating Disorders Examination Questionnaire; EDE-Q) and session by session review of progress (using the shorter ED-15). However, there is a paucity of high-quality instruments that have been validated for the full range of eating disorders among people with higher weight. Most eating disorder assessment tools have been developed and validated with predominantly low- or average-weight populations, and the language they use and concepts they measure, therefore present potential for stigmatisation and minimisation of pathology with higher weight (e.g., EDE-Q Item 11 of the shape concern subscale asks Have you felt fat?; and this is only considered an indicator of psychopathology in individuals of low weight). Health professionals are therefore advised to be aware of the limitations of these instruments and available to answer clarification questions in the context of a therapeutic interview. Also, the subscale scores can still be computed on the EDE-Q provided at least half the items for the particular subscale are completed which would allow item 11 to be skipped. Moreover, most validation studies for assessment measures have been conducted in predominately White female populations and therefore may not account for variations in eating practices seen in culturally and/or gender diverse samples.

Provided in Table 4 is a review of instruments recommended for use with people of higher weight. Please note that these instruments are not necessarily the most widely used nor the most frequently recommended for assessment of eating pathology in people with lower weight. Table 4 presents tools that have the most robust evidence for sensitivity, specificity and low risk of stigmatisation in the assessment of eating disorders for people with higher weight. More detailed information is provided in Additional file 1: Appendix D: Table of screening instruments.Footnote 7

Assessment of anorexia nervosa and dietary restrictionFootnote 8

The use of the broader ICD-11 diagnosis of anorexia nervosa without weight criterion (as is used in this guideline) is encouraged.

For detailed information regarding anorexia nervosa see Box 10. For the assessment of anorexia nervosa among people with higher weight, it is recommended that the EDE-Q (see "Assessment Instruments" for considerations regarding inappropriate items) , is used to examine restriction, with additional questions about total and recent weight loss [19].

Box 10 Anorexia nervosa (without low weight)

Dietary restriction may be used by a person to assist in emotion regulation, or in response to experiences of weight stigma and discrimination, without weight loss, especially where restriction leads to loss of control or binge eating. For the assessment of restriction without weight loss, additional scales such as the Dutch Eating Behaviour Questionnaire [DEBQ; examines emotional, external, restraint eating; 152], or the Modified Weight Bias Internalisation Scale (WBIS-M), may also be useful.

Assessment of binge or loss of control eating

Although the EDE-Q is a suitable assessment tool for eating disorders among people with higher weight (see "Assessment Instruments" for considerations regarding inappropriate items), it is known that the EDE-Q measurement of binge eating and compensatory behaviours is less reliable in this population. This is because the items that pick up on frequency of loss of c ontrol eating do not contribute towards the global EDE-Q score. If binge eating or loss of control is indicated in EDE-Q items-13–15, then it is optimal to also administer the Binge Eating Scale, as this latter measure provides a better examination of behavioural indicators and distress associated with binge eating. A loss of control overeating instrument may also be used, e.g., the Loss of Control over Eating Scales LOCES [LOCES; 153. Where ‘binge’ eating appears present without loss of control, the emotional and external eating subscales of the DEBQ [152] may also be useful, especially because it has been validated in a wide range of languages.

Another form of eating associated with loss of control is grazing [154] for which brief instruments have been developed [155,156,157] although to our knowledge these have not been validated in people with a high BMI.

Assessment of exercise

Assessment of exercise among people with eating disorders is either by self-report instrument or clinical interview. A recent systematic review identified two validated instruments specifically developed for people with eating disorders, namely the Compulsive Exercise Test and the Exercise and Eating Disorders [158]. Exercise may also be assessed objectively with an accelerometer or similar, but this is not recommended clinical practice and may be triggering for people with higher weight as these are frequently used in weight loss programs.

Assessment of muscle dysmorphia

Muscle dysmorphia is currently characterised in the DSM-5 as a specifier of body dysmorphic disorder and with obsessive–compulsive and related disorders. Although some research has suggested muscle dysmorphia is a subtype of body dysmorphia [159], other research suggests strong similarities with anorexia nervosa, where pathological concern with muscle gain replaces pathological concern with weight loss [160]. Recent research suggests muscle dysmorphia may have validity for a stand-alone diagnosis [161].

Individuals engaging in muscle building can have very high BMI due to high muscle density but low adiposity. They are at high-risk of a wide range of disordered eating behaviours [162], and use of anabolic steroids [163, 164]. For the assessment of muscle dysmorphia, the Muscle Appearance Satisfaction Scale (MASS), the Muscle Dysmorphia Questionnaire (MQDMDQ), the Muscle Dysmorphic Inventory (MDI) and the Muscle Dysmorphic Disorder Inventory (MDDI) are recommended.

Avoidant restrictive food intake disorder (ARFID)

ARFID is a newly described eating disorder and occurs across the weight spectrum. People living in larger bodies may experience ARFID and should be assessed and managed in the same way as for people not living in a larger body. The nine-item Avoidant/Restrictive Food Intake Disorder Screen (NIAS) is an assessment instrument which have been developed for adults [165]. The Child Food Neophobia Scale (CFNS) is a good psychometric measure of food avoidance in children [166].

Children and adolescents with higher weight

A recent study of adolescents in New South Wales [117] found that eating disorders were more likely to be experienced by adolescents who had a BMI percentile higher than those in the lower/average weight range. Further, adolescents who met criteria for bulimia nervosa, BED, anorexia nervosa (without low weight), subthreshold bulimia nervosa, or UFED had significantly greater odds of reporting high BMI, as compared to adolescents without these disorders. Younger adolescents (Grades 7–8; 13–14 years) were as likely to experience eating disorders as older adolescents (Grades 11–12; 17–18 years), though the distribution of diagnoses among these groups was different (with older adolescents significantly more likely to meet criteria for bulimia nervosa or BED). No effects of migrant or socioeconomic status were found on the likelihood of meeting criteria for any current eating disorders when controlling for age, gender and BMI percentile.

The Youth EDE-Q [YEDE-Q; 144] has been validated among adolescents with higher weight and includes age-appropriate language. The YEDE-Q is therefore recommended for evaluating eating disorder features in adolescents with higher weight.

Diabetes and eating disorders

Whilst the link between type 1 diabetes and low weight anorexia nervosa is well documented, there is a dearth of literature around anorexia nervosa (without low weight) and type 1 diabetes. Adolescents with type 1 diabetes who are of higher weight are at greater risk of disordered eating than peers with type 1 diabetes but not high weight [167]. Age, diabetes duration, cultural background, family structure, insulin regimen, daily insulin dose, or glycated haemoglobin A1c concentration have not been found to be associated with risk of onset disordered eating in adolescents with type 1 diabetes, but gender and BMI have. However, high glycated haemoglobin A1c may be a marker for insulin misuse and other harmful behaviours.

Among individuals with type 2 diabetes, the prevalence of eating disorders has been estimated to be between 6.5 and 9.0% [168]. There are more therapeutic options in the management of type 2 diabetes, with many people utilising non-insulin therapies, some of which are weight-neutral (metformin and dipeptidyl-peptidase 4 inhibitors) or promote weight loss (glucagon-like peptide 1 agonists or sodium-glucose transport protein 2 inhibitors) compared to agents that promote weight gain (insulin, Sulphonylureas and Pioglitazone). The selection of medication should be made on the basis of optimising blood sugar regulation in the long-term.

Two specific instruments have been developed for screening for eating disorders among individuals with diabetes: the Disordered Eating in Diabetes—Revised [DEPS-R; 169] and modified SCOFF [mSCOFF; 170]. However, because of issues with the validity and reliability of the SCOFF for people of higher weight, the DEPS-R is the recommended instrument, particularly in type 1 diabetes [see also 171]. This is because the DEPS-R has different psychometric properties according to whether the person under examination has type 1 diabetes requiring insulin, versus type 2 diabetes. Alternatively, use of the single question ‘I take less insulin than I should’ has been identified as potentially important for detecting eating disorder symptomology in people with diabetes who are using insulin [132].

Weight stigma

It is acknowledged that health professionals, because they are humans who are part of society and because of their socialisation as health professionals are likely to hold both implicit and explicit bias towards people with higher weight. The Academy of Eating Disorders recommends that all health professionals evaluate their own weight stigma with an online tool [172]. While some people with eating disorders may experience improved health with weight loss, to appropriately assess and treat people with eating disorders who are of higher weight, it is recommended that health professionals adopt a weight-inclusive or weight-neutral stance, advocating for increases in health behaviours and decreases in disordered eating, instead of a focus on weight loss, which can be perceived as inherently weight stigmatising [for a detailed analysis of how weight stigma can generate stress, disordered eating and further weight gain, see 9, 173]. To examine levels of internalised weight bias in people of higher weight, the Modified Weight Bias Internalisation scale [WBIS-M; 174] may be used to document links with eating disorder psychopathology.

Management overview

The major treatment approaches for all eating disorders have been outlined in “Current status of treatment and outcomes for all eating disorders” section. The following sections address treatment recommendations (see Tables 5, 6, 7, 8, 9, 10, 11) specifically for people with an eating disorder who are of higher weight. Treatment encompasses, but is not limited to psychological, pharmacological, nutritional and activity interventions. For all, it is important that management addresses all aspects of an eating disorder and thus will be, for the majority of people, multidisciplinary and requiring practitioners to work together as a formal or ‘virtual’ team through interprofessional collaborative practice (ICP) with each clinician practicing within the scope of their profession. ICP occurs when healthcare workers from different professional backgrounds work alongside the person experiencing the health condition, their supports, and communities to deliver collaborative care underpinned by teamwork, effective communication, and shared values [177]. This is recognised consistently throughout international and national guidelines and practice standards [48, 49, 178].

Table 5 Recommendation for the management of eating disorders for people with higher weight: management overview
Table 6 Recommendations for the management of eating disorders for people with higher weight: psychological therapy for adults
Table 7 Recommendations for the management of eating disorders for people with higher weight: psychological therapy for children and adolescents
Table 8 Recommendations for the management of eating disorders for people with higher weight: pharmacotherapy
Table 9 Recommendations for the management of eating disorders for people with higher weight: physical activity
Table 10 Recommendations for the management of eating disorders for people with higher weight: family and other interventions for adults, adolescents and children
Table 11 Recommendations for the management of eating disorders for people with higher weight: nutrition and medical management

Psychological therapy

Psychological therapy for adults

Evidence overview

At this time, there is no evidence to suggest that recommended evidence-based psychological treatments for eating disorders in adults of various weights (described in “Current status of treatment and outcomes for all eating disorders” section and in Table 2) are not appropriate for people of higher weight, however it is possible that they may benefit from adaptations or additions.

These psychological treatments include:

  • Cognitive behaviour therapy-enhanced (CBT-E), interpersonal psychotherapy (IPT) and dialectical behaviour therapy (DBT) for adults with bulimia nervosa or BED

  • Cognitive behaviour therapy (CBT), Maudsley model of anorexia nervosa treatment for adults (MANTRA), specialist supportive clinical management (SSCM) and focal psychodynamic therapy (FPT) for anorexia nervosa (without low weight)

Other approaches (e.g., BWLI) have been used for people with disorders characterised by recurrent binge eating, however these approaches are discussed only as they relate to their evidence for adults with an eating disorder and not as primary treatments for the eating disorder.

For this guideline specific research was sought for RCTs examining psychological treatments for eating disorders in adults with higher weight. A systematic review (Brennan et al. in preparation) has informed the majority of the literature presented in this guideline. A number of psychological treatments for eating disorders have been evaluated in RCTs specifically for the treatment of binge-eating disorder in adults with higher weight. These include CBT, IPT and DBT. Most of these interventions have been tested in group formats.

However, a major gap in research evidence is that RCTs in this population are nearly all confined to studies including participants with a diagnosis of BED. In particular, there were no RCTs examining the treatment of anorexia nervosa (without low weight) in people with higher weight. A further limitation was that the primary aims of most RCTs included in this review were to examine the effect of interventions on binge eating behaviours and weight. That is, higher body weight was positioned as (alongside binge eating) the therapeutic target, rather than body distress, pathological eating behaviours or eating disorder recovery. Thus, there is a need for measurements of a broader range of eating disorder outcomes (e.g., eating disorder psychopathology such as dietary restriction, body image dissatisfaction and self-induced vomiting), other psychosocial outcomes (e.g., quality of life, depression), and thorough assessment of potential harms. Follow-up in the longer term was also lacking. Further, the majority of trials of psychological interventions for people with BED (with the exception of CBT-E compared to another psychological intervention) are regarded as of low to very low quality due to high risk of bias in published reviews [e.g., 49].

Cognitive behaviour therapy (CBT)

CBT is the most frequently examined psychological intervention for eating disorders in adults with higher weight. Compared to wait list control groups, CBT has been shown to result in improvements in eating disorders symptoms [179,180,181]. CBT has also been shown to improve some body image aspects of eating disorder psychopathology (e.g., drive for thinness, body image dissatisfaction, eating concern, shape concern) relative to wait list control [182]. One study has investigated the impacts of involving spouses in CBT intervention. This did not impact on binge eating and it was associated with increased restraint [181]. CBT has been most commonly compared to BWLI and these studies are discussed below.

Brief and guided self-help CBT

Guided self-help (gsh)Footnote 9 interventions have also been trialled. CBTgsh resulted in greater improvement in binge eating than BWLgsh [183]. However, CBTgsh did not improve either binge eating relative to usual care [i.e. participants’ standard individual care from primary care physician; 184] or placebo [185]. One study comparing brief CBT comprised of 6-sessions delivered over 3 or 6 weeks demonstrated similar reductions in binge eating severity and frequency in both conditions [186]. Further, CBTgsh has been evaluated and found to be effective in reducing binge eating and other symptoms in many RCTs for people with binge-eating disorder where the majority of participants are at a higher weight [see 49, pp. 620–22].

Other psychological interventions

Other psychological interventions that have demonstrated improvements in eating disorder symptoms relative to wait list control include behavioural activation [187], and DBT [188, 189]. In one RCT, DBT also resulted in reduced binge eating behaviours and cognitions control after a 10-week intervention compared to a wait list [189].

To our knowledge, there is one study of a weight-inclusive therapy. Gaudiani [190] has reported an open case series of 12 individuals (92% women, mean age 36.7 years, SD = 6.8) with data extracted from electronic medical records. All were perceived as living in a larger body with high levels of eating disorder symptoms and low levels of intuitive eating. Eating disorder symptoms, intuitive eating and other psychological and physical health measures all significantly improved at follow-up. Notably, body weight was not measured during therapy or reported as an outcome as this is inconsistent with the treatment [190]. Systematic reviews have also found neutral or weightinclusive approaches such as HAES to be associated with improvements in eating behaviours (i.e., reduced cognitive restraint, disinhibition and binge eating) in people of a higher body weight [24, 25].

Other psychological treatments have also been compared to CBT. The one study comparing DBT to CBT reported no between group differences in eating disorder psychopathology at post-treatment, but the CBT group demonstrated greater improvements at follow-up. In addition, the CBT group demonstrated greater improvements in binge eating post-treatment, but no differences between treatments at follow-up [191]. One study comparing CBT to IPT demonstrated that both treatments resulted in comparable improvements in binge eating frequency and cessation post-treatment, and while there were minor increases in binge eating frequency at 12-month follow-up, both groups continued to demonstrate reductions in binge eating compared to pre-treatment. Both groups demonstrated reductions in pathological dietary restraint, CBT had larger effects post-treatment, but groups were equivalent at 12-month follow-up [192]. Hilbert et al. [193], have reported effects which were sustained in the longer-term, up to four years. A further study compared IPT, BWLI and CBTgsh and found that post-treatment all treatments produced improvements in binge eating frequency and cessation, and eating, shape and weight concerns, but that at 2-year follow-up IPT and CBTgsh resulted in greater binge eating remission rates, and BWLI resulted in greater cognitive restraint [78]. For people with a higher frequency of binge-eating, IPT appeared to be more effective than CBTgsh and BWLI [78].

Adapted treatments including CBT and BWLI for eating disorders characterised by recurrent binge eating

Psychological interventions have been most often compared to or used consecutively with BWLI. BWLI however aims to both reduce binge eating and elicit weight loss [see Box 2; 194]. While BWLI and CBT share some common characteristics (e.g., self-monitoring, use of behavioural strategies to reduce binge eating episodes) the primary goal of CBT is treatment of the eating disorder, and restraint is considered a maintaining factor and therefore a target of CBT interventions is the reduction of restraint. Furthermore, most RCTs have found CBT to be more effective than BWLI in improving eating disorder symptoms (e.g., binge eating) and in some cases achieving remission of binge eating [195,196,197]. However, in some studies there is no difference between treatments at 6-months [199], or 12-month follow-up [196], and in other studies, between-group differences are greater at 6-month follow-up [197, 198].

Only a few studies comparing CBT and BWLI have measured other eating disorder psychopathology such as body image concerns [194]. One found that CBT and BWLI resulted in similar improvements in eating, weight and shape concern [196]. Conversely, Grilo et al. [197] found that neither of these treatments produced an effect on these variables nor on restraint. One further study found BWLI to increase restraint relative to CBT [78], and another that CBT resulted in greater improvements in eating, weight and shape concern, but not restraint, relative to BWLI [202]. Only one study comparing CBT to BWLI has conducted long-term follow-up. At post treatment, CBT resulted in greater improvements in binge eating frequency and BED diagnosis [196]. However, there were no differences between groups at 6-year follow-up [199].

Other studies have evaluated sequential CBT and BWLI. For example, in one study participants who responded to CBT (i.e., improved eating disorder symptoms) were then offered BWLI while those who did not respond to CBT were offered IPT. The responders offered BWLI intervention demonstrated further improvements in binge eating and further weight loss, while the non-responders offered IPT demonstrated increased binge eating and small increases in weight [179]. In a second study, participants received either CBT, BWLI, or CBT followed by BWLI. There were no differences in binge eating remission between groups post treatment, but at 6-month follow-up the CBT alone group demonstrated significantly greater binge eating remission than BWLI alone or in combination with CBT [197].

A recent study compared BWLI to a stepped care model in which non-responders to BWLI were stepped up to CBTgsh. Both conditions demonstrated significant improvements in binge eating remission and frequency, with no difference between groups [200].

One RCT has tested an integrated BWLI with CBT-E in a transdiagnostic group with BN, BED and OSFED [201].Footnote 10 There were significant within group reductions in eating disorder symptoms but only one between group difference for main eating disorder psychopathology outcomes. This was an increased binge eating remission rate with the integrated intervention at one year compared to CBT-E. Secondary outcomes are yet to be published [206]. Cooper, Calugi and Dalle Grave [203] have also proposed an integrated treatment but this is as yet untested.

A systematic review of mindfulness-based interventions for people of higher weight found that mindfulness-based interventions resulted in a significant decrease of binge-eating disorder symptoms, when compared with control [204]. However this was an exploratory analysis due to the limited number (i.e., three) of studies available.

CBT and other dietary and non-dietary interventions

CBT in combination with dietary interventions, such as low calorie diets (LCDs) or nutritional counselling, has not demonstrated advantages over CBT alone with regards to eating disorder symptoms [205, 206]. In contrast, combining CBT with inpatient treatment for obesity has been shown to improve binge eating episodes, relative to inpatient treatment alone, at 12-month follow-up [207]. BWLI has also been compared to non-dieting interventions (promoting improvements in health behaviours and body image without intentional weight loss). Both resulted in improvements in binge eating severity [208]. More recent weight neutral or weight-inclusive approaches, such as HAES, have shown improvements in eating behaviours (i.e., reduced cognitive restraint, disinhibition and binge eating) however such interventions have no published evidence to date in people with eating disorders [24, 25].

Clinical considerations

There are some important issues specific to the treatment of people with eating disorders who are of higher weight that clinicians should be aware of.

Approaches for people with anorexia nervosa/restrictive eating disorders

Resumption of menses has been identified as an important treatment goal for females with restrictive eating disorders as it is a factor contributing to improved bone mineral density [209]. Restoration to pre-morbid weight, even if this is at a relatively high BMI, may achieve the most complete and long-lasting recovery [210]. However, research on weight restoration for anorexia nervosa among people living in larger bodies is currently lacking but has been noted as a priority for future research.

The value of in-session collaborative weighing

Evidence-based psychological therapies for eating disorders all stress the importance of in-session weighing. This is to monitor weight for safety reasons (e.g., in the case of anorexia nervosa and related disorders to make sure the person is restoring weight and/or not losing weight) as well as for the purpose of achieving cognitive change. However, when working with people with eating disorders who are of higher weight, the value of in-session weighing should be carefully considered, and the benefits evaluated against the risks of any possible negative consequences. For some people with higher weight, in-session weighing is recommended but options such as blind weighing can be considered. Again, this issue should be raised by the therapist and discussed openly with the individual before treatment begins. Where malnutrition is suspected (for example after prolonged dietary restriction or significant weight loss, regardless of current body weight) or there are medical co-morbidities present, a dietitian and a general practitioner should be closely involved in care and may use weight change as a marker of nutritional status. However, as above, weight change can be monitored without the person being aware of their weight if that is their preference.

Weight stigma

As highlighted in “Weight stigma” section ‘weight stigma’, therapists working with people experiencing eating disorders who are of higher weight need to be aware of the negative effects of weight stigma, and that fact that they, themselves, may be influenced by weight stigma which may make it more difficult to focus treatment on the person’s eating disorder rather than on their weight. Further training and supervision by a skilled clinician in this area may be helpful.

Psychological therapy for children and adolescents

Evidence overview

There is no evidence to suggest that current evidence-based treatments for eating disorders in children and adolescents are not appropriate for people with higher weight. As outlined in “Current status of treatment and outcomes for all eating disorders” section FBT is the first line treatment for anorexia nervosa and bulimia nervosa for this age group, with second line treatments for anorexia nervosa being adolescent focused therapy (AFT) and CBT-E. CBT-E is also considered a second line treatment for bulimia nervosa. However, guidelines vary as to how strongly these second line treatments are recommended [49, 52]. For BED, adult treatments are recommended [49] and for ARFID there is no recommendation, but CBT is noted as promising [52]. As noted earlier, an evidence-base for specific psychological interventions or modifications to current evidence-based treatments for those with higher weight does not exist.

Clinical considerations

Modification of current evidence-based treatment for young people with and eating disorder and who are of higher weight is not yet indicated and treatment directives such as weighing the person experiencing the eating disorder in session should be followed. However, clinicians should proceed with sensitivity and judgement mindful of the potential for increasing shame and the impact of weight stigma and how this may impact on the young person’s and family experience. Some aspects of public health campaigns focussing on reducing childhood obesity (e.g. weighing of children in school) may trigger the development of an eating disorder in vulnerable young people. A common clinical impression from parents is the lack of recognition they can receive for their child’s difficulties and the delay this creates in receiving help. Young people on the other hand, often feel a sense of failure to be ‘seen’ as sick enough because of their weight. These and other related experiences should be recognised and integrated into the young person and family’s treatment to improve engagement.


Evidence overview

There are no medications developed for the treatment of people experiencing an eating disorder who are of higher weight where the primary outcome is improvement in eating disorder symptoms and/or behaviours. There are also no medications recommended in current general guidelines [48, 49] as first line in the treatment of an eating disorder. Whilst RCTs have found evidence of efficacy for some medications, for example, SSRIs particularly in people with BED or bulimia nervosa, effects are not sustained when the medication is withdrawn [48]. There are two groups of medications that are, however relevant to the scope of this guideline:

  1. 1.

    Medications that may be used for people with eating disorders. These are not recommended as ‘first-line’; they are most often used as adjunctive treatments.

  2. 2.

    Medications used to reduce appetite with potential to impact on eating disorder treatment.

It is also important to acknowledge that research in the use of medications in BED has been biased towards participants of whom either all or a very high proportion were people with higher weight. For example, in the NICE [49] guidelines all reported RCTs of pharmacological therapies in BED are of participants with a high BMI (> mean 30). Covertly or overtly, weight loss/maintenance in these trials is often presumed to be a positive treatment outcome.

Furthermore as we have noted medications are most often used as adjunctive treatments where they may enhance the efficacy of the psychological therapy however the present state of evidence is insufficient to recommend routine use in addition to psychological therapies.

Medications that may be used for people with eating disorders (see also Table 3)


This is a stimulant approved in Australia for treatment of BED. It is not approved for appetite suppression but has this effect. It is cautioned and is a relative contraindication in people with histories of substance use disorder and/or who are in the underweight range, in a state of weight loss or weight suppression. This is particularly true for people with past or current anorexia nervosa and some people with bulimia nervosa. Most efficacy trials have included a majority of people with a high BMI.


The majority of evidence for efficacy of antidepressants for people of a high BMI and an eating disorder is confined to BED and is of low to very low quality. They are inferior to CBT, and there is insufficient evidence they will enhance CBT or other psychological therapies. Relative risk for remission is 1.39 (0.92–209) in four studies to 12 months [49; Table 275]. Most evidence is for fluoxetine (up to 80 mg per day in BED; 60 mg per day in bulimia nervosa). Antidepressants may be considered for bulimia nervosa and BED where there is co-occurring depression or difficulties accessing psychological therapy. Antidepressants may reduce appetite in the short-term and/or be associated with reduced appetite in the longer term.


There is limited evidence for the use of topiramate in bulimia nervosa and BED. It is poorly tolerated with several adverse effects including weight loss, sedation and neurological symptoms [211]. One RCT of lamotrigine [212] in people with BED with higher weight reported a very high placebo response, similar to the active drug effect for binge eating.

Antipsychotics/mood regulating agents

All antipsychotics and mood regulating agents, but particularly second-generation medications such as olanzapine, may cause increased appetite, weight gain and exacerbate conditions associated with a high BMI such as metabolic syndrome and type 2 diabetes [213]. They also have a range of other problematic adverse effects such as sedation. When prescribed for a person with higher weight, one that is least likely to impact on appetite should be considered [214]. If there is severe weight gain, a change in antipsychotic/mood regulating agent should be considered as people may develop an eating disorder or exacerbation of eating disorder symptoms as a consequence.

Other agents

Atomoxetine is a selective norepinephrine reuptake inhibitor. Evidence in eating disorders is limited to one trial in BED where it was associated with binge eating reduction [215]. Similarly Armodafinil, a psychostimulant has been found in one trial of BED to reduce binge eating [216]. Finally, dasotraline, a new agent with dual dopamine and noradrenaline reuptake inhibition, has been found in two RCTs to reduce binge eating in people with BED [217, 218]. It also reduced appetite in people with higher weight [219]. None of these agents are approved for use in eating disorders in Australia.

Medications used to reduce appetite

The weight loss medication orlistat has been trialled in people with BED who are of higher weight but it has poor tolerability and there have been reports of its abuse in people with bulimia nervosa [220]. It has not been approved for use in BED in Australia. Medications such as metformin, insulin and semaglutide may alter food consumption and consideration of this, and potential for non-prescribed use needs to be applied in the care of a person living with a higher body weight and an eating disorder.

Physical activity

Evidence overview

While there has been much research on exercise interventions for people of higher weight, few studies directly examine physical activity in the treatment of eating disorders among people with higher weight. However, a range of physical and psychological benefits (e.g., improved self-perception, body image and mood) have been found in studies involving structured and tailored exercise interventions in eating disorder populations. Such exercise is commonly part of a broader lifestyle, BWLI or LCD intervention and may take place in the workplace, where people spend a large portion of their time. It includes the implementation of walking routes, team exercise classes, improvements in cafeteria/vending machine options and team psychoeducation [221]. It is likely that these programs vary greatly in their weight-centrism and potential to reinforce weight stigma. As this literature does not directly assess or refer to underlying eating disorder psychopathology caution is needed when translating such findings to eating disorder populations where exercise can become compulsive and used in an attempt to compensate for binge eating episodes. Meta-analyses have consistently found that exercising for predominately weight and shape reasons is likely to be associated with the onset and/or exacerbation of an eating disorder [222,223,224].

Levine et al. [225] looked at the effects of a 6-month exercise intervention in women with BMI > 30 and BED and found significant reductions in binge eating symptomatology in the treatment group compared with control, but no difference in effect on depressive symptomatology. Pendleton et al. [226] trialled exercise-augmented CBT in BED and also found significant reductions in binge eating symptomatology post-treatment. McIver et al. [227] found a yoga intervention significantly reduced self-reported binge eating in higher weight individuals as compared with a wait list control group who did not improve on any measure at post-test.

Clinical considerations

The literature has been evaluated in conjunction with clinical expertise to inform this guideline, and further research is needed to build a solid evidence-base. Primary treatment goals in this population should be psychotherapeutic and focus on self-acceptance and the development of a healthy relationship with exercise [228]. Emphasis should be placed on the physical and mental health benefits of regularly engaging in exercise [229], and more importantly, improvements in self-perception and positive wellbeing [230, 231] rather than a narrow focus on weight. Whether conducted with a normative or general eating disorder population, research consistently demonstrates multicomponent approaches including psychoeducation to be more broadly effective for improving physical and psychological health than behavioural changes alone [232]. What constitutes ‘effective’ will also depend on the individual and their goals. Exercise recommendations rarely consider current fitness levels, impaired mobility, or existing mental health concerns, such as eating disorders. Wherever possible, people with an eating disorder and are of higher weight should be engaged with a multidisciplinary team and any exercise or physical activity program should be closely monitored by a trained eating disorder and exercise professional, begin at an appropriate intensity and increase slowly over time in a graded fashion [228; see Box 11].

Box 11 Exercise in eating disorder treatment

Notably, clinical judgement should be utilised when dealing with vulnerable populations. For people with eating disorders exercise can be pathological or unhelpful in nature or frequency, thus exercise interventions for those people with higher weight need to take a different approach. People with higher weight may face additional challenges when attempting to implement exercise interventions due to current and/or previous experiences of weight stigma, prejudice and discrimination. Notably, exposure to exercise environments (such as gyms) very often involve exposure to weight stigmatising environments.

Family and other interventions for adults, adolescents and children

Evidence overview

The evidence-base for family interventions specific to people with an eating disorder who are of higher weight is extremely limited and no interventions developed for children and adolescents with eating disorders note any specific treatment adjustments for young people with higher weight. Further, none of the adult family interventions reported above (“Current status of treatment and outcomes for all eating disorders” section) specifically address or recommend an augmentation for people with eating disorders who are of higher weight.

People with anorexia nervosa (without low weight) may live, or have previously lived in a larger body. While FBT (see “Context” section, Table 2) was initially developed for people with eating disorders who are in an underweight range, there is some evidence for its application to individuals with anorexia nervosa (without low weight) without augmentation of the model [234]. However, a recent qualitative study of practitioners applying FBT to people with anorexia nervosa (without low weight) identified a lack of clarity on appropriate weight targets, the use of the weight chart in treatment and difficulty activating urgency in the parents [235]. These are all critical aspects of FBT for anorexia nervosa.

There is a body of literature around the negative effects of weight/shape and eating conversations, from familial, peer and other sources, for children and adolescents. Amongst many psychological consequences is an increased risk of eating problems [236, 237].

Clinical considerations

Clinicians should implement evidence-based treatment interventions for people with eating disorders who are of higher weight as recommended and continue to involve families in treatment. At the least, psychoeducation of families and supports are needed. This would include emphasising that nutrition is critical, providing information about what constitutes normal eating, and the way in which malnutrition impacts the brain and makes body distortion/fear of weight gain worse. Nutritional recovery often leads to weight gain, regardless of the person’s initial starting weight. Similarly, nutritional recovery commonly results in improved cognitive function, although improvements in eating disorder thinking often lags behind other changes. It is ideal to deliver psychoeducation on psychosocial impacts of an eating disorder when in a larger body. This may include how families manage their own weight stigma and conflicting advice from health professionals regarding the desirability of weight loss.

Structured support from family/supports to facilitate regular and adequate eating will assist with eating disorder cognitions and returning a normal eating pattern. This may include the responsibility of purchasing of food, preparing of meals, and support at mealtime. Families should be encouraged to check in with their own assumptions about body shape and size so their loved one can focus on recovering from the eating disorder, rather than on a fear of returning to or maintaining larger body size. Families should be encouraged to use body neutral and body positive talk. Health professionals reflecting on their own use of body negative talk and overvaluation of shape and size is important. Changing our own language and thoughts can model body image acceptance and a focus on health in recovery.

Families should be encouraged and supported to develop distress tolerance skills for both themselves and the person with the eating disorder rather than using disordered eating behaviours to reduce distress. Encouraging families and supports to consider social media usage in the home and supporting media literacy in the eating disorder affected person is also likely to be helpful. Evidence-based resources include Mental Health First Aid, including online resources, and eating disorder specific training.

It is important to note that families are also expected to be active in the second line individual treatments (CBT-E and AFT) discussed earlier. While parents are not present for every session their role is defined and participation critical. Similarly, the inclusion of parents in any emerging treatment for children and adolescents is going to be essential given the importance of both the relationship and family context. IPT for BED as discussed in “Psychological: first line” section is an exemplar of a novel treatment also being delivered in a family-based format [238].

Nutritional and medical management

Evidence overview

The research evidence for nutrition care for people with an eating disorder who are of higher weight is covered above in the sections on BWLI, CBT and other dietary interventions, and exercise. There is no evidence to support any dietary intervention as stand-alone care for treatment of an eating disorder. Nutritional assessment and management of nutritional care in larger-bodied people with eating disorders is best provided with the support of a dietitian.

Clinical considerations

The nutritional and medical management of person with an eating disorder who are of higher weight must address both the eating disorder and any other health needs of the individual. This may include nutritional complications of the eating disorder, and the nutritional needs of physical and mental health co-occurring condition. A priority is the nutritional management of medical conditions such as type 1 diabetes, with awareness that an eating disorder may complicate dietary management.


Addressing malnutrition is essential for preventing life-threatening and longer-term complications in those with a restrictive or other eating disorders [19, 239]. Malnutrition is generally defined as a BMI < 18.5 kg/m2 or unintentional weight loss of ≥ 5% with evidence of suboptimal intake resulting in subcutaneous fat loss and/or muscle wasting regardless of BMI [240]. However, intentional weight loss, or being in a state of ‘weight suppression’ (i.e., a discrepancy between one’s highest adult weight and current weight), should not preclude a diagnosis of malnutrition in someone with an eating disorder, and identifying malnutrition beyond current weight, with assessment of percentage of weight loss is recommended by the American Academy of Paediatrics, American Society for Parenteral and Enteral Nutrition, Academy of Nutrition and Dietetics and the Society for Adolescent Health and Medicine [209, 241, 242]. However, identification and assessment are only the first steps in the nutritional rehabilitation process required to reverse malnutrition.

There are numerous clinical guidelines outlining the best evidence-based strategies for treating malnutrition and improving dietary quality [243], which may help guide the nutritional interventions for malnourished people with an eating disorder who are of higher weight. However, it is important to ensure that nutritional rehabilitation not only addresses immediate nutritional needs to prevent further weight loss, but also the body’s need for physical repair of any damage incurred during dietary restriction and other eating disordered behaviours resulting in malnutrition. A person’s body weight may need to increase to allow for this physical repair and restoration. This may be difficult for the person with an eating disorder to accept when their sense of identity is closely linked to their appearance, and they have been striving to lose weight. They also will encounter, and be distressed by, the negative consequences and stigma (perceived or actual) of a higher weight.

Micronutrient deficiencies

People with higher weight may have micronutrient deficiencies (e.g., zinc, iron, vitamin D, B-group vitamins, etc.) due to low diet quality and potentially reduced bioavailability [244,245,246]. Moreover, eating disorders may potentially result in micronutrient abnormalities or deficiencies as a result of dietary restriction and eating disorder behaviours (e.g., vomiting) leading to medical complications.

Other medical problems

While higher weight is associated with a multitude of medical and psychological conditions, this section deals with the management of medical conditions in people with both an eating disorder and with higher weight. Eating disordered behaviours in people with higher weight may also lead to a range of medical complications that require intervention. As previously mentioned, people of any weight with BED are at risk of medical complications such as type 2 diabetes, hypertension and dyslipidaemia [47]. These conditions often require specific dietary restriction and modification. Although traditional dietetic interventions for people with higher weight with such medical conditions have promoted the primary goal of specific dietary modification for weight loss [247249], these effects appear short-term, and may bring unhelpful consequences such as weight regain, binge eating, body dissatisfaction, eating disorders and low self-esteem 250, 251. Further, health gains may be achieved with improved diet quality alone [252, 253].Footnote 11

Nutritional guidance on management of such medical complications therefore needs to be aware of language and avoid messaging that can reinforce poor self-worth, feelings of failure and stigmatisation, which can all contribute to worsening eating disorder behaviours rather than reducing the medical complications. Individualised nutrition counselling and dietary adaptations to manage medical co-occurring conditions are important. This may include non-weight loss focussed dietary approaches and HAES approaches, which incorporate directly targeting unconditional body shape and size acceptance, and encourage physical activity and eating for well-being, including eating according to appetite, decreasing vulnerability to external stimuli and coping with emotional eating. A systematic review of randomised and non-randomised studies examining HAES interventions for management of BMI suggests that HAES, focusing on more comprehensive health outcomes rather than weight loss alone, may be effective for improving some cardiovascular outcomes, but further studies examining the effect on blood glucose and blood pressure are needed [24].

The presence of binge eating, purging and other eating disorder behaviours complicates the management of diabetes. Goebel-Fabbri [258] has written a practical guide to management of eating disorders and type 1 diabetes, some of which is also relevant for management of type 2 diabetes. A clinical guideline for disordered eating and eating disorders in adults with type 1 diabetes (aged 16 years and over) produced by Queensland Health is also available [259]. Polycystic ovary syndrome (PCOS) is also associated with an increased risk of disordered eating [260, 261] and care needs to be taken not to exacerbate body image issues and eating disorders in this group of women [262].

In the case of bone health, although people with higher weight are thought to have higher bone mineral density (BMD), they appear to have an increased risk of fractures at some sites [263]. If severe dietary restriction and malnutrition is layered on top of this, leading to inadequate calcium intake and potentially a fall in oestrogen in females, this may place the individual at an increased risk of fractures. Current research suggests adults with anorexia nervosa (without low weight) have significant bone deficits, while adolescents with anorexia nervosa (without low weight) have BMD scores higher than adolescents with anorexia nervosa who are underweight [264], with their BMD potentially protected by their premorbid higher weight.Further, in atypical anorexia nervosa, lack of current low weight or amenorrhoea does not prevent reduced vertebral strength [265], and should be considered as a potential concern in all individuals with an eating disorder who have a history of severe dietary restriction. However, findings have been inconsistent [e.g., 266].

Bariatric surgery

It is important to assess for an eating disorder in people with higher weight attending for bariatric surgery assessment, as the prevalence is high [267]. People with a history of eating disorders also often plan to undergo bariatric and/or cosmetic surgery [268]. Additionally, although binge eating and psychological conditions like anxiety and depression may improve in the short-term following bariatric surgery, they may restart over the longer term [269, 270]. Continuing psychological support may improve outcomes in the longer-term from bariatric surgery [271]. However, the data are quite mixed and most point to the need for an improved understanding of who will develop loss of control eating after surgery as opposed to prior to surgery.

Other psychiatric therapy for co-occurring conditions (e.g., bipolar disorder, psychosis)

Both BED and bipolar spectrum disorders are frequent co-occurring conditions in people with higher weight, and experiencing both BED and bipolar disorder concurrently is associated with more severe eating behaviours and psychopathology [272]. Furthermore, it is suggested that approximately 10% of people with schizophrenia have BED [273]. People with such psychiatric co-occurring conditions often require antipsychotic medication which is associated with rapid weight gain and metabolic abnormalities as detailed earlier [274,275,276]. These medications are known to increase appetite, decrease satiety and increase cravings for sweet foods and drinks, as well as contribute to disordered eating habits, such as only eating one main meal each day [277, 278]. Mood stabilisers (e.g., lithium) and anticonvulsants (e.g., valproate) can also have weight gain effects [276]. In people with an eating disorder who are of higher weight who are also taking antipsychotic medication, it is important to be aware of the risk of onset of disordered eating and eating disorders in this context.

Cultural considerations

Evidence-based knowledge of cultural considerations in the management of eating disorders is in its infancy. To our knowledge there are no studies that specifically addresses cultural considerations for the treatment of eating disorders for people with higher weight. The following paragraphs are derived from research pertaining to cultural considerations for the treatment of eating disorders (at any weight) as well as lived experience and clinical expertise. The below groups were chosen as salient groups that are under-represented in the eating disorders literature and treatment services within the Australian context, however such considerations may be relevant for similarly under-represented and disadvantaged groups across the international context. See a recent systematic review by Acle et al. [279] for empirically derived guidance on how to effectively address culture in eating disorder treatment among racial/ethnic minorities. A lived experience perspective is provided in Box 12.

Box 12 A lived experience perspective: cultural considerations

Men with eating disorders

Historically perceived as disorders of women, eating disorders can affect people of any gender. While there has been an under representation of males in eating disorder research [280], it is estimated that one third of people reporting eating disorder behaviours in the community are male [281]. Males account for approximately 30% of people with bulimia nervosa, 57% of people with BED, 55–77% of people with OSFED [subtype-dependent; 282] and 67% of ARFID [283].

In comparison with women, men are more likely to have a history of higher weight prior to their eating disorder, accompanied by weight-related bullying [236, 284]. In addition to weight stigma and the stigma associated with having a mental illness, males may experience stigma associated with having a ‘female’ disorder which may present as a barrier to seeking and engaging in treatment [33, 285]. Men also experience a later age of onset [281] and higher rates of co-occurring psychiatric conditions [286]. Despite this, research shows that health professionals are less likely to recognise eating disorder behaviours in males as a mental health problem, and this less likely to offer treatment [280, 287].

In Westernised society, the majority of males report desiring a lean muscular physique [288, 289] as opposed to a ‘thinner’ physique often desired by women [290]. This pursuit of a muscularity may manifest in a wide range of eating disorders behaviours including misuse of anabolic steroids [163, 164].

While men can experience all eating disorder diagnoses, some differences in eating disorder psychopathology have been noted across genders. Men are less likely to report a loss of control over eating, despite having similar rates of objective binge eating to women and are more likely to engage in compulsive exercise for emotion regulation [291]. The management approaches described throughout this guideline are not gender-specific, however health professionals may need to hold additional considerations in mind when working with men such as the importance of exploring and challenging ‘masculine’ concepts of strength, power and control for greater treatment engagement [292]. Clinicians are also encouraged to be attuned to how men express and communicate (often gendered) emotions including distress, anger, grief, irritability, anxiety and sadness. For additional information on considerations for psychological therapy when working with men with eating disorders see Bunnell [293].

Aboriginal and Torres Strait Islander peoples

Owing to the limited evidence for the treatment of eating disorders for Aboriginal and Torres Strait Islander people, health professionals working with people experiencing eating disorders and their families, should apply caution when applying this guideline to Indigenous peoples and recognise there may be a need to customise or tailor current treatment and communication approaches to accommodate their culturally diverse needs, resources and expectations.

It is suggested that health professionals refrain from using clinical language and overreliance of health literature in awareness that some Indigenous peoples have lower literacy levels and/or English as a second language, lower health literacy, and lower mental health literacy than non-Indigenous Australians.

A clinical yarning approach [see 294] could help mitigate any potential barriers with establishing therapeutic rapport, service engagement and possible referral pathways. When making recommendations for treatment, health professionals should be aware that Indigenous peoples often face multiple access barriers (e.g., cost, transport, limited range of service for rural and remote communities) especially when needing to access multiple and ongoing health care as is required for eating disorder treatment.

Health professionals are also encouraged to conceptualise eating disorders from the perspective of social emotional wellbeing [see 295]. Social emotional wellbeing is phrase and holistic concept of health unique to Aboriginal and Torres Strait Islander peoples and distinguishes the understanding of mental health disorders from the medical orientated, euro-centric conceptualisation of mental health and treatment. As such, standard nutrition guidance may not be suitable for Aboriginal and Torres Strait Islander People who are accustomed to living off the country or are experiencing high rates of food insecurity [296]. Additionally, it is important for health professionals to understand that the shame experienced by some people with disordered eating behaviours may vary across cultures and a tailored understanding of shame in the context of Aboriginal and Torres Strait Islander people is necessary.

It is also recommended that health professionals practice and provide trauma informed care (see Box 3) due to the ongoing and intergenerational trauma, grief and loss consequential to colonisation and its continual impact on contemporary Aboriginal and Torres Strait Islander peoples [297]. Practicing cultural reflexivity (i.e., critically examining one’s own attitudes, values and biases) is a step towards cultural competency. Working in true partnership with Aboriginal and Torres Strait Islander people (i.e., acknowledging the person experiencing the eating disorder, their family and community as equally experts in the process) and collaborating with Aboriginal-led medical and community services or Aboriginal allied health professionals may foster cultural safety and improve engagement [297].

Finally, health professionals are encouraged to broaden their perspective of what constitutes an Indigenous person’s support system which may often involve input from Elders, community members, extended family and friends. It is also important to explore the role of Traditional Healers and bush medicines people, where and if appropriate.

LGBTIQA+ Footnote 12individuals

Research on the prevalence of eating disorders in gender and sexual minority people is limited, however, emerging research suggests higher rates of eating disorders in LGBTIQA  (lesbian, gay, bisexual, transgender, gender diverse, intersex, queer, asexual and questioning) people compared to their heterosexual and cisgender peers [298, 299]. Health professionals may need to hold in mind additional considerations and tailor aspects of management and communication when working with LGBTIQA+ people with eating disorder who are of higher weight.

Body image dissatisfaction is a core symptom and stressor for sexual and gender minorities and a significant risk factor for the development of an eating disorder [300]. This is especially true for the transgender population where higher levels of incongruence between biological and assigned sex and gender identity are related to higher levels of body image dissatisfaction [301]. Clinicians should explicitly seek consent to physical examine a person’s body and have an awareness of the potential distress related to physical examinations, especially when gender dysphoria is present.

Practicing trauma-informed care (see Box 3) is of particular importance when working with LGBTIQA+ people as research suggests this population experiences higher rates of adverse events compared to the general population [302,303,304]. People from sexual and gender minorities may face additional stressors including ‘minority stress’, i.e., identity-based stress experienced by members of disadvantaged social groups, over and above the general life stressors experiences by all members of society [305] as well as ‘intra-minority stress’ i.e., stress derived from within the LGBTIQA+ community [306]. LGBTIQA+ people with eating disorders who are of higher weight endure ‘double stigma’ (i.e., weight stigma as well as the stigma from being in a minority group) and associated prejudice and discrimination [307, 308].

Research suggests that 40% of transgender people with an eating disorder did not disclose their gender identity to their clinicians due to fears (based on past experiences with health professionals) of being ignored, stigmatized and/or discriminated against [309]. It is important that health professionals foster a sense of safety by being gender affirmative and do not make assumptions about a person’s gender or sexual identity. This may include asking the person experiencing an eating disorder about their pronouns and seeking consent before disclosing their gender or sexual identity to other health professionals, family members and/or supports. Using gender neutral language when discussing management with people experiencing eating disorders (e.g., swapping the terms ‘breast’ for the term ‘chest’; ‘motherhood’ for ‘parenthood’; and ‘breastfeeding’ for ‘nursing’) and may help validate a person’s gender identity and foster a safe healthcare environment. Health professionals are encouraged not to make assumptions about people’s body image and/or body image distress as stereotypes of an ‘ideal body/shape/weight’ may vary across LGBTIQA+ cultures.

Finally, clinicians are encouraged to expand their perspective of what constitutes a family and support system to include ‘chosen and created families’ (i.e., non-nuclear supports) who may provide vital support throughout the treatment journey for people with an eating disorder who are of higher weight. While this also applies to both heterosexual and cisgender people, it is of particular importance for LGBTIA+ people, who, when compared to heterosexual cisgender people are more likely to live alone, less likely to have children and more likely to be estranged from their biological family [310].


A summary of key recommendations9 is provided in Table 12.

Table 12 Summary of key recommendations


In conclusion, this guideline have compiled a series of recommendation for the approach and care of people with eating disorders who have higher body weight. This guideline has been written from the perspective of the adverse effects of weight stigma and the complexity of causes of eating disorders across people of all sizes. The readers are referred to other literature for management of specific medical and other psychological disorders that are often experienced by people with an eating disorder who are living in a larger body.

Availability of data and materials

Not applicable.


  1. Up to the inclusion of bulimia nervosa in DSM-III in 1980 [1], the only eating disorder that was recognised was anorexia nervosa, historically associated with a low BMI.

  2. When BMI is used in this guideline it is broadly based on the World Health Organization (WHO) BMI categories for adults over 20 years old, i.e.: BMI < 18.5 is underweight; BMI 18.5–24.9 is adequate weight; BMI ≥ 25 is overweight; and, BMI ≥ 30 is in an obese weight range [26].

  3. OSFED includes presentations that do not meet the full criteria for any of the disorders in the feeding and eating disorders diagnostic class including: atypical anorexia nervosa; bulimia nervosa (of low frequency and/or limited duration); binge eating disorder (of low frequency and/or limited duration); purging disorder; and night eating syndrome.

  4. There are other syndromes such as orthorexia nervosa, emotional overeating and food addiction which are also disorders of feeding and/or eating. However, they are not considered in the present guideline as they are outside the major intentional diagnostic schemes. Similarly, diabulimia is not a diagnosable eating disorder, but rather a term used to denote insulin misuse as a weight control behaviour and/or compensation for binge eating episodes in people with diabetes and an eating disorder.

  5. This section outlines the current status of treatment and outcomes for all eating disorders, including for people who have low weight anorexia nervosa.

  6. NESARC-III (i.e. third wave of National Epidemiologic Survey on Alcohol and Related Conditions) is the largest epidemiologic household survey of US adults that assessed eating disorders.

  7. Note. Table 4 does not contain a comprehensive list of all instruments that may be used in the assessment of people with eating disorders who are of higher weight, but as stated, we selected those with the most ro bust evidence. There are many instruments (e.g., Emotional Eating Scale for children and adolescents [EES-C; 138]; Eating in the Absence of Hunger Questionnaire for children and adolescents [EAH-C; 139]; Questionnaire of Eating and Weight Patterns—adolescent version [QEWP-A; 140]; Children’s Eating Attitude Test [ChEAT; [141, 142]); and, the Repe titive Eating Questionnaire (Rep (eat)-Q [157])   that m ay also be used in this context.

  8. Restriction is a reduction in dietary intake, eating less food than your body requires. Restraint is the intention to restrict.

  9. Guided self-help (self-help material with clinician guidance) is distinct from pure self-help (self-help material only).

  10. Palavras et al., [201] was not included in the Brennan et al., review (manuscript in preparation) as the primary outcome was weight loss. It did however investigate a broad range of outcome including quality of life, eatin g dis order psychopathology and physical health stature and met the inclusion criteria for the scope of this guideline.

  11. It should be noted that in long-term follow-up of interventions aimed to reduce disorders associated with high weight, improvements have been reported in body dissatisfaction and binge eating frequency [254257], However, these are studies of high weight disorder and not of people with both an eating disorder and with high weight for whom the results cannot be directly applied and are out of scope for this guideline.

  12. It is acknowledged that there are differences across issues of gender identity compared with sexual identity, however for ease of reference, both are included in this section. It is also acknowledged that some terminology is contested and language in this area is evolving.



Avoidant/restrictive food intake disorder


Adolescent focused therapy


Binge-eating disorder


Body mass index


Behavioural weight loss interventions


Cognitive behaviour therapy


Cognitive behaviour therapy-enhanced


Dialectical behaviour therapy


Eye movement desensitisation and reprocessing


Family based treatment


Focal psychodynamic therapy


Guided self-help


Health at every size®


Interprofessional collaborative practice


Interpersonal psychotherapy


Low calorie diet


Lesbian, gay, bisexual, transgender, gender diverse, intersex, queer, asexual and questioning


Maudsley model of anorexia nervosa treatment for adults


National Health and Medical Research Council


National Institute for Health and Care Excellence


National Eating Disorders Collaboration


Other feeding or eating disorder


Polycystic ovary syndrome


Preferred reporting items for systematic and meta-analysis


Randomised controlled trial


Repetitive transcranial magnetic stimulation


Specialist supportive clinical management


Unspecified feeding or eating disorder


World Health Organization


  1. American Psychiatric Association. Diagnostic and statistical manual of mental disorders. 3rd ed. 1980.

  2. Da Luz F, Sainsbury A, Mannan H, Touyz S, Mitchison D, Hay P. Prevalence of obesity and comorbid eating disorder behaviors in South Australia from 1995 to 2015. Int J Obes. 2017;41(7):1148–53.

    Article  Google Scholar 

  3. Hart LM, Ferreira KB, Ambwani S, Gibson EB, Austin SB. Developing expert consensus on how to address weight stigma in public health research and practice: a Delphi study. Stigma Health. 2020;6:179–89.

    Google Scholar 

  4. Academy for Eating Disorders Nutrition Working Group. Guidebook for nutrition treatment of eating disorders. 2020.

  5. Wagner A, Butt M, Rigby A. Internalized weight bias in patients presenting for bariatric surgery. Eat Behav. 2020;39:1–20.

    Article  Google Scholar 

  6. Lawson JL, LeCates A, Ivezaj V, Lydecker J, Grilo CM. Internalized weight bias and loss-of-control eating following bariatric surgery. Eating Disorders. 2020;29(6):1–14.

  7. Lee KM, Arriola-Sanchez L, Lumeng JC, Gearhardt A, Tomiyama AJ. Weight Stigma by Association Among Parents of Children With Obesity: A Randomized Trial. Acad Pediatr. 2021;22(5):754–60.

  8. Vartanian LR, Porter AM. Weight stigma and eating behavior: a review of the literature. Appetite. 2016;102:3–14.

    Article  PubMed  Google Scholar 

  9. Tomiyama AJ. Weight stigma is stressful. A review of evidence for the cyclic obesity/weight-based stigma model. Appetite. 2014;82:8–15.

    Article  PubMed  Google Scholar 

  10. Ajibewa T. A psychobiobehavioral expansion of the cyclic obesity/weight-based stigma (COBWEBS) model in adolescents with overweight and obesity [dissertation]. University of Michigan. 2021.

  11. O’Brien KS, Latner JD, Puhl RM, Vartanian LR, Giles C, Griva K, et al. The relationship between weight stigma and eating behavior is explained by weight bias internalization and psychological distress. Appetite. 2016;102:70–6.

    Article  PubMed  Google Scholar 

  12. Schvey NA, Roberto CA, White MA. Clinical correlates of the weight bias internalization scale in overweight adults with binge and purge behaviours. Adv Eat Disord Theory Res Pract. 2013;1(3):213–23.

    Google Scholar 

  13. Durso LE, Latner JD, White MA, Masheb RM, Blomquist KK, Morgan PT, et al. Internalized weight bias in obese patients with binge eating disorder: associations with eating disturbances and psychological functioning. Int J Eat Disord. 2012;45(3):423–7.

    Article  PubMed  Google Scholar 

  14. Almeida L, Savoy S, Boxer P. The role of weight stigmatization in cumulative risk for binge eating. J Clin Psychol. 2011;67(3):278–92.

    Article  PubMed  Google Scholar 

  15. Phelan SM, Burgess DJ, Yeazel MW, Hellerstedt WL, Griffin JM, van Ryn M. Impact of weight bias and stigma on quality of care and outcomes for patients with obesity. Obes Rev. 2015;16(4):319–26.

    Article  PubMed  PubMed Central  Google Scholar 

  16. Mensinger JL, Tylka TL, Calamari ME. Mechanisms underlying weight status and healthcare avoidance in women: a study of weight stigma, body-related shame and guilt, and healthcare stress. Body Image. 2018;25:139–47.

    Article  PubMed  Google Scholar 

  17. Stunkard AJ. Pain of obesity. Palo Alto: Bull Publishing Company; 1976.

    Google Scholar 

  18. Appolinario JC, Sichieri R, Lopes CS, Moraes CE, Veiga dGV, Freitas S, et al. Correlates and impact of DSM-5 binge eating disorder, bulimia nervosa and recurrent binge eating: a representative population survey in a middle-income country. Soc Psychiatry Psychiatr Epidemiol. 2022;19:1–3.

  19. Whitelaw M, Lee KJ, Gilbertson H, Sawyer SM. Predictors of complications in anorexia nervosa and atypical anorexia nervosa: Degree of underweight or extent and recency of weight loss? J Adolesc Health. 2018;63(6):717–23.

    Article  PubMed  Google Scholar 

  20. Sawyer SM, Whitelaw M, Le Grange D, Yeo M, Hughes EK. Physical and psychological morbidity in adolescents with atypical anorexia nervosa. Pediatrics. 2016;137(4): e20154080.

  21. Butryn ML, Webb V, Wadden TA. Behavioral treatment of obesity. Psychiatry Clin N Am. 2011;34(4):841–59.

    Article  Google Scholar 

  22. Dombrowski SU, Knittle K, Avenell A, Araújo-Soares V, Sniehotta FF. Long term maintenance of weight loss with non-surgical interventions in obese adults: systematic review and meta-analyses of randomised controlled trials. Br Med J. 2014;348:1–12.

    Article  Google Scholar 

  23. Dansinger ML, Tatsioni A, Wong JB, Chung M, Balk EM. Meta-analysis: the effect of dietary counseling for weight loss. Ann Intern Med. 2007;147(1):41–50.

    Article  PubMed  Google Scholar 

  24. Ulian MD, Aburad L, Da Silva Oliveira MS, Poppe ACM, Sabatini F, Perez I, et al. Effects of health at every size® interventions on health-related outcomes of people with overweight and obesity: a systematic review. Obes Rev. 2018;19(12):1659–66.

    Article  PubMed  Google Scholar 

  25. Dugmore JA, Winten CG, Niven HE, Bauer J. Effects of weight-neutral approaches compared with traditional weight-loss approaches on behavioral, physical, and psychological health outcomes: A systematic review and meta-analysis. Nutr Rev. 2020;78(1):39–55.

    Article  PubMed  Google Scholar 

  26. World Health Organization Europe. Body mass index—BMI 2021.

  27. Zembic A, Eckel N, Stefan N, Baudry J, Schulze MB. An empirically derived definition of metabolically healthy obesity based on risk of cardiovascular and total mortality. J Am Med Assoc Netw Open. 2021;4(5):1–14.

    Google Scholar 

  28. Nagata JM, Golden NH. New US Preventive Services Task Force Recommendations on Screening for Eating Disorders. JAMA Internal Medicine. 2022;182(5):471–3.

  29. World Health Organization. International Classification of Diseases-ICD. Eleventh edition 2019.

  30. Galmiche M, Déchelotte P, Lambert G, Tavolacci MP. Prevalence of eating disorders over the 2000–2018 period: a systematic literature review. Am J Clin Nutr. 2019;109(5):1402–13.

    Article  PubMed  Google Scholar 

  31. Hay P, Mitchison D, Collado AEL, González-Chica DA, Stocks N, Touyz S. Burden and health-related quality of life of eating disorders, including avoidant/restrictive food intake disorder (ARFID), in the Australian population. J Eat Disord. 2017;5(1):21–30.

    Article  PubMed  PubMed Central  Google Scholar 

  32. Qian J, Wu Y, Liu F, Zhu Y, Jin H, Zhang H, et al. An update on the prevalence of eating disorders in the general population: a systematic review and meta-analysis. Eating and Weight Disorders-Studies on Anorexia, Bulimia and Obesity. 2021;27:415–28.

  33. Thapliyal P, Hay PJ. Treatment experiences of males with an eating disorder: a systematic review of qualitative studies. Transl Dev Psychiatry. 2014;2(1):1–9.

    Google Scholar 

  34. Calzo JP, Blashill AJ, Brown TA, Argenal RL. Eating disorders and disordered weight and shape control behaviors in sexual minority populations. Curr Psychiatry Rep. 2017;19(8):1–10.

    Article  Google Scholar 

  35. Mulders-Jones B, Mitchison D, Girosi F, Hay P. Socioeconomic correlates of eating disorder symptoms in an Australian population-based sample. PLoS ONE. 2017;12(1):1–17.

    Article  Google Scholar 

  36. Cheah SL, Jackson E, Touyz S, Hay P. Prevalence of eating disorder is lower in migrants than in the Australian-born population. Eat Behav. 2020;37:1–8.

    Article  Google Scholar 

  37. Volpe U, Tortorella A, Manchia M, Monteleone AM, Albert U, Monteleone P. Eating disorders: what age at onset? Psychiatry Res. 2016;238:225–7.

    Article  PubMed  Google Scholar 

  38. Burt A, Mitchison D, Doyle K, Hay P. Eating disorders amongst Aboriginal and Torres Strait Islander Australians: a scoping review. J Eat Disord. 2020;8(1):1–8.

    Article  Google Scholar 

  39. Hoek HW. Review of the worldwide epidemiology of eating disorders. Curr Opin Psychiatry. 2016;29(6):336–9.

    Article  PubMed  Google Scholar 

  40. Solmi M, Köhler CA, Stubbs B, Koyanagi A, Bortolato B, Monaco F, et al. Environmental risk factors and nonpharmacological and nonsurgical interventions for obesity: An umbrella review of meta‐analyses of cohort studies and randomized controlled trials. Eur J Clin Invest. 2018;48(12):e12982.

  41. Solmi M, Radua J, Stubbs B, Ricca V, Moretti D, Busatta D, et al. Risk factors for eating disorders: an umbrella review of published meta-analyses. Braz J Psychiatry. 2020;43(3):314–23.

    Article  PubMed Central  Google Scholar 

  42. Hailes HP, Yu R, Danese A, Fazel S. Long-term outcomes of childhood sexual abuse: an umbrella review. Lancet Psychiatry. 2019;6(10):830–9.

    Article  PubMed  PubMed Central  Google Scholar 

  43. Matthews A, Peterson CM, Mitan L. Adolescent males with atypical anorexia nervosa and premorbid obesity: three case reports. Eat Weight Disord Stud Anorex Bulim Obes. 2019;24(5):963–7.

    Article  Google Scholar 

  44. Schaumberg K, Welch E, Breithaupt L, Hübel C, Baker JH, Munn-Chernoff MA, et al. The science behind the academy for eating disorders’ nine truths about eating disorders. Eur Eat Disord Rev. 2017;25(6):432–50.

    Article  PubMed  PubMed Central  Google Scholar 

  45. Arcelus J, Mitchell AJ, Wales J, Nielsen S. Mortality rates in patients with anorexia nervosa and other eating disorders: a meta-analysis of 36 studies. Arch Gen Psychiatry. 2011;68(7):724–31.

    Article  PubMed  Google Scholar 

  46. Santomauro DF, Melen S, Mitchison D, Vos T, Whiteford H, Ferrari AJ. The hidden burden of eating disorders: An extension of estimates from the Global Burden of Disease Study 2019. Lancet Psychiatry. 2021;8(4):320–8.

    Article  PubMed  PubMed Central  Google Scholar 

  47. Udo T, Grilo CM. Psychiatric and medical correlates of DSM-5 eating disorders in a nationally representative sample of adults in the United States. Int J Eat Disord. 2019;52(1):42–50.

    Article  PubMed  Google Scholar 

  48. Hilbert A, Hoek HW, Schmidt R. Evidence-based clinical guidelines for eating disorders: International comparison. Curr Opin Psychiatry. 2017;30(6):423–37.

    Article  PubMed  PubMed Central  Google Scholar 

  49. National Institute for Health and Care Excellence. Eating disorders: Recognition and treatment. Version 2.0. Full guideline. United Kingdom: the National Institute of Health and Care Excellence. 2017.

  50. Eisler I, Lock J, Le Grange D. Family based treatmemnts for adolescents with anorexia nervosa: single-family and multifamily approaches. In: Grilo CM, Mitchell JE, editors. The treatment of eating disorders: a clinical handbook. New York: Guilford Press; 2010. p. 150–74.

    Google Scholar 

  51. Lock J, Le Grange D. Treatment manual for anorexia nervosa: a family based approach. 2nd ed. New York: Guilford Press; 2013.

    Google Scholar 

  52. Couturier J, Isserlin L, Norris M, Spettigue W, Brouwers M, Kimber M, et al. Canadian practice guidelines for the treatment of children and adolescents with eating disorders. J Eat Disord. 2020;8(4):1–804.

    Google Scholar 

  53. Rienecke RD. Family-based treatment of eating disorders in adolescents: Current insights. Adolesc Health Med Ther. 2017;8:69–79.

    PubMed  PubMed Central  Google Scholar 

  54. Halvorsen I, Reas DL, Nilsen JV, Rø Ø. Naturalistic outcome of family-based inpatient treatment for adolescents with anorexia nervosa. Eur Eat Disord Rev. 2018;26(2):141–5.

    Article  PubMed  Google Scholar 

  55. Matthews A, Peterson CM, Peugh J, Mitan L. An intensive family-based treatment guided intervention for medically hospitalized youth with anorexia nervosa: parental self-efficacy and weight-related outcomes. Eur Eat Disord Rev. 2019;27(1):67–75.

    Article  PubMed  Google Scholar 

  56. Simic M, Stewart CS, Eisler I, Baudinet J, Hunt K, O’Brien J, et al. Intensive treatment program (ITP): a case series service evaluation of the effectiveness of day patient treatment for adolescents with a restrictive eating disorder. Int J Eat Disord. 2018;51(11):1261–9.

    Article  PubMed  Google Scholar 

  57. Dimitropoulos G, Freeman VE, Allemang B, Couturier J, McVey G, Lock J, et al. Family-based treatment with transition age youth with anorexia nervosa: a qualitative summary of application in clinical practice. J Eat Disord. 2015;3(1):1–13.

    Article  PubMed  PubMed Central  Google Scholar 

  58. Gorrell S, Le Grange D. Update on treatments for adolescent bulimia nervosa. Child Adolesc Psychiatr Clin. 2019;28(4):537–47.

    Article  Google Scholar 

  59. Lock J. Family-based treatment for avoidant/restrictive food intake disorder. 1st ed. London: Routledge; 2021.

    Book  Google Scholar 

  60. Thomas JJ, Becker KR, Kuhnle MC, Jo JH, Harshman SG, Wons OB, et al. Cognitive-behavioral therapy for avoidant/restrictive food intake disorder: Feasibility, acceptability, and proof-of-concept for children and adolescents. Int J Eat Disord. 2020;53(10):1636–46.

    Article  PubMed  PubMed Central  Google Scholar 

  61. Tanofsky-Kraff M, Wilfley DE, Young JF, Mufson L, Yanovski SZ, Glasofer DR, et al. A pilot study of interpersonal psychotherapy for preventing excess weight gain in adolescent girls at-risk for obesity. Int J Eat Disord. 2010;43(8):701–6.

    Article  PubMed  PubMed Central  Google Scholar 

  62. Hilbert A, Petroff D, Neuhaus P, Schmidt R. Cognitive-behavioral therapy for adolescents with an age-adapted diagnosis of binge-eating disorder: a randomized clinical trial. Psychother Psychosom. 2020;89(1):51–4.

    Article  PubMed  Google Scholar 

  63. Mazzeo SE, Lydecker J, Harney M, Palmberg AA, Kelly NR, Gow RW, et al. Development and preliminary effectiveness of an innovative treatment for binge eating in racially diverse adolescent girls. Eat Behav. 2016;22:199–205.

    Article  PubMed  PubMed Central  Google Scholar 

  64. Linardon J, Fairburn CG, Fitzsimmons-Craft EE, Wilfley DE, Brennan L. The empirical status of the third-wave behaviour therapies for the treatment of eating disorders: a systematic review. Clin Psychol Rev. 2017;58:125–40.

    Article  PubMed  Google Scholar 

  65. Treasure J, Rhind C, Macdonald P, Todd G. Collaborative care: the new Maudsley model. Eat Disord. 2015;23(4):366–76.

    Article  PubMed  Google Scholar 

  66. Fleming C, Le Brocque R, Healy K. How are families included in the treatment of adults affected by eating disorders? A scoping review. Int J Eat Disord. 2020;54(3):244–79.

    Article  PubMed  Google Scholar 

  67. Baucom DH, Kirby JS, Fischer MS, Baucom BR, Hamer R, Bulik CM. Findings from a couple-based open trial for adult anorexia nervosa. J Fam Psychol. 2017;31(5):584–91.

    Article  PubMed  PubMed Central  Google Scholar 

  68. Nyman-Carlsson E, Norring C, Engström I, Gustafsson SA, Lindberg K, Paulson-Karlsson G, et al. Individual cognitive behavioral therapy and combined family/individual therapy for young adults with Anorexia nervosa: a randomized controlled trial. Psychother Res. 2020;30(8):1011–25.

    Article  PubMed  Google Scholar 

  69. Dimitropoulos G, Farquhar JC, Freeman VE, Colton PA, Olmsted MP. Pilot study comparing multi-family therapy to single family therapy for adults with anorexia nervosa in an intensive eating disorder program. Eur Eat Disord Rev. 2015;23(4):294–303.

    Article  PubMed  Google Scholar 

  70. Tantillo M, McGraw JS, Lavigne HM, Brasch J, Le Grange D. A pilot study of multifamily therapy group for young adults with anorexia nervosa: reconnecting for recovery. Int J Eat Disord. 2019;52(8):950–5.

    Article  PubMed  Google Scholar 

  71. Wierenga CE, Hill L, Knatz Peck S, McCray J, Greathouse L, Peterson D, et al. The acceptability, feasibility, and possible benefits of a neurobiologically-informed 5-day multifamily treatment for adults with anorexia nervosa. Int J Eat Disord. 2018;51(8):863–9.

    Article  PubMed  Google Scholar 

  72. Runfola CD, Kirby JS, Baucom DH, Fischer MS, Baucom BR, Matherne CE, et al. A pilot open trial of UNITE-BED: A couple-based intervention for binge-eating disorder. Int J Eat Disord. 2018;51(9):1107–12.

    Article  PubMed  Google Scholar 

  73. Le Grange D, Eckhardt S, Dalle Grave R, Crosby RD, Peterson CB, Keery H, et al. Enhanced cognitive-behavior therapy and family-based treatment for adolescents with an eating disorder: a non-randomized effectiveness trial. Psychol Med. 2020; 1–11.

  74. Dalle Grave R, Eckhardt S, Calugi S, Le Grange D. A conceptual comparison of family-based treatment and enhanced cognitive behavior therapy in the treatment of adolescents with eating disorders. J Eat Disord. 2019;7(1):1–9.

    Article  Google Scholar 

  75. Lock J. Adolescent-focused therapy for anorexia nervosa: A developmental approach. New York: Guilford Publications; 2020.

    Google Scholar 

  76. Eisler I, Simic M, Hodsoll J, Asen E, Berelowitz M, Connan F, et al. A pragmatic randomised multi-centre trial of multifamily and single family therapy for adolescent anorexia nervosa. BMC Psychiatry. 2016;16(1):1–14.

    Article  Google Scholar 

  77. Lafrance Robinson A, Dolhanty J, Stillar A, Henderson K, Mayman S. Emotion-focused family therapy for eating disorders across the lifespan: a pilot study of a 2-day transdiagnostic intervention for parents. Clin Psychol Psychother. 2016;23(1):14–23.

    Article  PubMed  Google Scholar 

  78. Wilson GT, Wilfley DE, Agras WS, Bryson SW. Psychological treatments of binge eating disorder. Arch Gen Psychiatry. 2010;67(1):94–101.

    Article  PubMed  PubMed Central  Google Scholar 

  79. Martenstyn JA, Touyz S, Maguire S. Treatment of compulsive exercise in eating disorders and muscle dysmorphia: protocol for a systematic review. J Eat Disord. 2021;9(1):1–7.

    Article  Google Scholar 

  80. Marcos YQ, Zarceño EL, López JAL. Effectiveness of exercise-based interventions in patients with anorexia nervosa: a systematic review. Eur Eat Disord Rev. 2020;29(1):3–19.

    Article  Google Scholar 

  81. Vancampfort D, Vanderlinden J, Hert MD, Soundy A, Adámkova M, Skjaerven LH, et al. A systematic review of physical therapy interventions for patients with anorexia and bulemia nervosa. Disabil Rehabil. 2014;36(8):628–34.

    Article  PubMed  Google Scholar 

  82. Zeeck A, Schlegel S, Jagau F, Lahmann C, Hartmann A. The Freiburg sport therapy program for eating disorders: a randomized controlled trial. J Eat Disord. 2020;8(1):31–43.

    Article  PubMed  PubMed Central  Google Scholar 

  83. Dittmer N, Voderholzer U, Mönch C, Cuntz U, Jacobi C, Schlegl S. Efficacy of a specialized group intervention for compulsive exercise in inpatients with anorexia nervosa: a randomized controlled trial. Psychother Psychosom. 2020;89(3):161–73.

    Article  PubMed  Google Scholar 

  84. Mathisen TF, Bratland-Sanda S, Rosenvinge JH, Friborg O, Pettersen G, Vrabel KA, et al. Treatment effects on compulsive exercise and physical activity in eating disorders. J Eat Disord. 2018;6(1):1–9.

    Article  Google Scholar 

  85. Ng LWC, Ng DP, Wong WP. Is supervised exercise training safe in patients with anorexia nervosa? A meta-analysis. Physiotherapy. 2013;99(1):1–11.

    Article  PubMed  Google Scholar 

  86. Moola FJ, Gairdner SE, Amara CE. Exercise in the care of patients with anorexia nervosa: a systematic review of the literature. Ment Health Phys Act. 2013;2(6):59–68.

    Article  Google Scholar 

  87. Hall PA, Vincent CM, Burhan AM. Non-invasive brain stimulation for food cravings, consumption, and disorders of eating: a review of methods, findings and controversies. Appetite. 2018;124:78–88.

    Article  PubMed  Google Scholar 

  88. Brewerton TD. An overview of trauma-informed care and practice for eating disorders. J Aggress Maltreat Trauma. 2019;28(4):445–62.

    Article  Google Scholar 

  89. Smyth JM, Heron KE, Wonderlich SA, Crosby RD, Thompson KM. The influence of reported trauma and adverse events on eating disturbance in young adults. Int J Eat Disord. 2008;41(3):195–202.

    Article  PubMed  Google Scholar 

  90. Berge JM, Loth K, Hanson C, Croll-Lampert J, Neumark-Sztainer D. Family life cycle transitions and the onset of eating disorders: a retrospective grounded theory approach. J Clin Nurs. 2012;21(9–10):1355–63.

    Article  PubMed  Google Scholar 

  91. Dansky BS, Brewerton TD, Kilpatrick DG, O’Neil PM. The national women’s study: Relationship of victimization and posttraumatic stress disorder to bulimia nervosa. Int J Eat Disord. 1997;21(3):213–28.

    Article  PubMed  Google Scholar 

  92. Molendijk M, Hoek H, Brewerton T, Elzinga B. Childhood maltreatment and eating disorder pathology: a systematic review and dose-response meta-analysis. Psychol Med. 2017;47(8):1402–16.

    Article  Google Scholar 

  93. Lian Q, Su Q, Li R, Elgar FJ, Liu Z, Zheng D. The association between chronic bullying victimization with weight status and body self-image: a cross-national study in 39 countries. PeerJ. 2018;6:1–16.

    Article  Google Scholar 

  94. Puhl RM, Peterson JL, Luedicke J. Weight-based victimization: bullying experiences of weight loss treatment–seeking youth. Pediatrics. 2013;131(1):1–9.

    Article  Google Scholar 

  95. Johns G, Taylor B, John A, Tan J. Current eating disorder healthcare services—the perspectives and experiences of individuals with eating disorders, their families and health professionals: systematic review and thematic synthesis. BJPsych Open. 2019;5(4):1–10.

    Article  Google Scholar 

  96. Brewerton TD. Eating disorders, trauma, and comorbidity: focus on PTSD. Eat Disord. 2007;15(4):285–304.

    Article  PubMed  Google Scholar 

  97. Brewerton TD. Eating disorders, victimization, and comorbidity: principles of treatment. Clinical handbook of eating disorders. Boca Raton: CRC Press; 2004. p. 535–72.

    Google Scholar 

  98. Trim JG, Galovski TE, Wagner A, Brewerton TD. Treating eating disorder-posttraumatic stress disorder patients: A synthesis of the literature and new treatment directions. In: Anderson LK, Murray SB, Kaye WH, editors. Clinical handbook of complex and atypical eating disorders Oxford University Press; 2018. p. 40–59.

  99. Friederich H-C, Wild B, Zipfel S, Schauenburg H, Herzog W. Anorexia nervosa: focal psychodynamic psychotherapy. Göttingen: Hogrefe; 2018.

    Book  Google Scholar 

  100. Tanofsky-Kraff M, Wilfley DE. Interpersonal psychotherapy for bulimia nervosa and binge-eating disorder. In: Grilo CM, Mitchell JE, editors. The treatment of eating disorders: a clinical handbook. Guildford Press: New York; 2010. p. 271–93.

    Google Scholar 

  101. Safer DL, Telch CF, Chen EY. Dialectical behavior therapy for binge eating and bulimia. New York: Guilford Press; 2009.

    Google Scholar 

  102. Wonderlich SA, Peterson CB, Crosby RD, Smith TL, Klein MH, Mitchell JE, et al. A randomized controlled comparison of integrative cognitive-affective therapy (ICAT) and enhanced cognitive-behavioral therapy (CBT-E) for bulimia nervosa. Psychol Med. 2014;44(3):543–53.

    Article  PubMed  Google Scholar 

  103. Udo T, Grilo CM. Physical activity levels and correlates in nationally representative sample of US adults with healthy weight, obesity, and binge-eating disorder. Int J Eat Disord. 2020;53(1):85–95.

    Article  PubMed  Google Scholar 

  104. Coffino JA, Udo T, Grilo CM. The significance of overvaluation of shape or weight in binge-eating disorder: Results from a national sample of US adults. Obesity. 2019;27(8):1367–71.

    Article  PubMed  Google Scholar 

  105. Olguin P, Fuentes M, Gabler G, Guerdjikova AI, Keck PE, McElroy SL. Medical comorbidity of binge eating disorder. Eat Weight Disord Stud Anorex Bulim Obes. 2017;22(1):13–26.

    Article  Google Scholar 

  106. Mehler PS, Rylander M. Bulimia nervosa–medical complications. J Eat Disord. 2015;3(1):1–5.

    Google Scholar 

  107. Handzlik-Orlik G, Holecki M, Orlik B, Wyleżoł M, Duława J. Nutrition management of the post–bariatric surgery patient. Nutr Clin Pract. 2015;30(3):383–92.

    Article  PubMed  Google Scholar 

  108. Cederholm T, Barazzoni R, Austin P, Ballmer P, Biolo G, Bischoff SC, et al. ESPEN guidelines on definitions and terminology of clinical nutrition. Clin Nutr. 2017;36(1):49–64.

    Article  PubMed  Google Scholar 

  109. Garber AK, Cheng J, Accurso EC, Adams SH, Buckelew SM, Kapphahn CJ, et al. Weight loss and illness severity in adolescents with atypical anorexia nervosa. Pediatrics. 2019;144(6):1–13.

    Article  Google Scholar 

  110. Tanofsky-Kraff M, Shomaker LB, Stern EA, Miller R, Sebring N, DellaValle D, et al. Children’s binge eating and development of metabolic syndrome. Int J Obes. 2012;36(7):956–62.

    Article  Google Scholar 

  111. Radin RM, Tanofsky-Kraff M, Shomaker LB, Kelly NR, Pickworth CK, Shank LM, et al. Metabolic characteristics of youth with loss of control eating. Eat Behav. 2015;19:86–9.

    Article  PubMed  PubMed Central  Google Scholar 

  112. Hay P, Chinn D, Forbes D, Madden S, Newton R, Sugenor L, et al. Royal Australian and New Zealand College of Psychiatrists clinical practice guidelines for the treatment of eating disorders. Aust N Z J Psychiatry. 2014;48(11):977–1008.

    Article  PubMed  Google Scholar 

  113. National Health and Medical Research Council. Guidelines for Guidelines Handbook: National Health and Medical Research Council; 2016.

  114. Chen Y, Yang K, Marušić A, Qaseem A, Meerpohl JJ, Flottorp S, et al. A reporting tool for practice guidelines in health care: the RIGHT statement. Ann Intern Med. 2017;166(2):128–32.

    Article  PubMed  Google Scholar 

  115. Brennan L, Mellody K, Hindle A, de La Piedad Garcia X, Hay P, Ralph AF, Byrne S. Treatment of eating disorders in individuals living in a larger body: a systematic review and meta analyses PROSPERO 2020 CRD42020179083.

  116. Aromataris E, Fernandez R, Godfrey CM, Holly C, Khalil H, Tungpunkom P. Summarizing systematic reviews: methodological development, conduct and reporting of an umbrella review approach. JBI Evid Implement. 2015;13(3):132–40.

    Google Scholar 

  117. Mitchison D, Mond J, Bussey K, Griffiths S, Trompeter N, Lonergan A, et al. DSM-5 full syndrome, other specified, and unspecified eating disorders in Australian adolescents: Prevalence and clinical significance. Psychol Med. 2020;50(6):981–90.

    Article  PubMed  Google Scholar 

  118. Hart LM, Granillo MT, Jorm AF, Paxton SJ. Unmet need for treatment in the eating disorders: a systematic review of eating disorder specific treatment seeking among community cases. Clin Psychol Rev. 2011;31(5):727–35.

    Article  PubMed  Google Scholar 

  119. Feltner C, Peat C, Reddy S, Riley S, Berkman N, Middleton JC, et al. Screening for eating disorders in adolescents and adults: evidence report and systematic review for the US Preventive Services Task Force. JAMA. 2022;327(11):1068–82.

    Article  PubMed  Google Scholar 

  120. US Preventetive Services Task Force. Screening for eating disorders in adolescents and adults: US Preventive Services Task Force recommendation statement. JAMA. 2022;327(11):1061–7.

    Article  Google Scholar 

  121. Sim LA, Lebow J, Billings M. Eating disorders in adolescents with a history of obesity. Pediatrics. 2013;132(4):1026–30.

    Article  Google Scholar 

  122. Heriseanu AI, Hay P, Touyz S. Grazing behaviour and associations with obesity, eating disorders, and health-related quality of life in the Australian population. Appetite. 2019;143:1–10.

    Article  Google Scholar 

  123. Cotton MA, Ball C, Robinson P. Four simple questions can help screen for eating disorders. J Gen Intern Med. 2003;18(1):53–6.

    Article  PubMed  PubMed Central  Google Scholar 

  124. American Psychiatric Association. Diagnostic and statistical manual of mental disorders. 5th ed. 2013.

  125. Wells KR, Jeacocke NA, Appaneal R, Smith HD, Vlahovich N, Burke LM, et al. The Australian Institute of Sport (AIS) and National Eating Disorders Collaboration (NEDC) position statement on disordered eating in high performance sport. Br J Sports Med. 2020;54(21):1247–58.

    Article  PubMed  Google Scholar 

  126. Leffler DA, Dennis M, George JBE, Kelly CP. The interaction between eating disorders and celiac disease: an exploration of 10 cases. Eur J Gastroenterol Hepatol. 2007;19(3):251–5.

    Article  PubMed  Google Scholar 

  127. Zickgraf HF, Hazzard VM, O’Connor SM. Food insecurity is associated with eating disorders independent of depression and anxiety: Findings from the 2020–2021 Healthy Minds Study. Int J Eat Disord. 2022;55(3):354–61.

  128. Leehr EJ, Krohmer K, Schag K, Dresler T, Zipfel S, Giel KE. Emotion regulation model in binge eating disorder and obesity—a systematic review. Neurosci Biobehav Rev. 2015;49:125–34.

    Article  PubMed  Google Scholar 

  129. Evans EH, Adamson AJ, Basterfield L, Le Couteur A, Reilly JK, Reilly JJ, et al. Risk factors for eating disorder symptoms at 12 years of age: a 6-year longitudinal cohort study. Appetite. 2017;108:12–20.

    Article  PubMed  PubMed Central  Google Scholar 

  130. Lavender JM, Brown TA, Murray SB. Men, muscles, and eating disorders: an overview of traditional and muscularity-oriented disordered eating. Curr Psychiatry Rep. 2017;19(6):1–7.

    Article  Google Scholar 

  131. García-Mayor RV, García-Soidán FJ. Eating disoders in type 2 diabetic people: Brief review. Diabetes Metab Syndr. 2017;11(3):221–4.

    Article  PubMed  Google Scholar 

  132. Goebel-Fabbri AE, Fikkan J, Franko DL, Pearson K, Anderson BJ, Weinger K. Insulin restriction and associated morbidity and mortality in women with type 1 diabetes. Diabetes Care. 2008;31(3):415–9.

    Article  PubMed  Google Scholar 

  133. Rodin G, Olmsted MP, Rydall AC, Maharaj SI, Colton PA, Jones JM, et al. Eating disorders in young women with type 1 diabetes mellitus. J Psychosom Res. 2002;53(4):943–9.

    Article  PubMed  Google Scholar 

  134. Reardon CL, Hainline B, Aron CM, Baron D, Baum AL, Bindra A, et al. Mental health in elite athletes: International Olympic Committee consensus statement (2019). Br J Sports Med. 2019;53(11):667–99.

    Article  PubMed  Google Scholar 

  135. Gladstone J. The skinny on BMI-based hiring: an assessment of the legality and effectiveness of Israel’s weight restriction law. Wash U Global Stud L Rev. 2016;15:495.

    Google Scholar 

  136. BMC. Eating disorder treatment practice standards 2020.

  137. Hurst K, Heruc G, Thornton C, Freeman J, Fursland A, Knight R, et al. ANZAED practice and training standards for mental health professionals providing eating disorder treatment. J Eat Disord. 2020;8(1):1–10.

    Article  Google Scholar 

  138. Tanofsky-Kraff M, Theim KR, Yanovski SZ, Bassett AM, Burns NP, Ranzenhofer LM, et al. Validation of the emotional eating scale adapted for use in children and adolescents (EES-C). Int J Eat Disord. 2007;40(3):232–40.

    Article  PubMed  PubMed Central  Google Scholar 

  139. Tanofsky-Kraff M, Ranzenhofer LM, Yanovski SZ, Schvey NA, Faith M, Gustafson J, et al. Psychometric properties of a new questionnaire to assess eating in the absence of hunger in children and adolescents. Appetite. 2008;51(1):148–55.

    Article  PubMed  PubMed Central  Google Scholar 

  140. Johnson WG, Grieve FG, Adams CD, Sandy J. Measuring binge eating in adolescents: adolescent and parent versions of the questionnaire of eating and weight patterns. Int J Eat Disord. 1999;26(3):301–14.

    Article  PubMed  Google Scholar 

  141. Maloney MJ, McGuire J, Daniels SR, Specker B. Dieting behavior and eating attitudes in children. Pediatrics. 1989;84(3):482–9.

    Article  PubMed  Google Scholar 

  142. Maloney MJ, McGUIRE JB, Daniels SR. Reliability testing of a children’s version of the Eating Attitude Test. J Am Acad Child Adolesc Psychiatry. 1988;27(5):541–3.

    Article  PubMed  Google Scholar 

  143. Fairburn CG. Eating Disorder Examination Questionnaire (EDE-Q 6.0). In: Fairburn CG, editor. Cognitive behaviour therapy and eating disorders. New York, NY: Guilford Press; 2008.

  144. Goldschmidt AB, Doyle AC, Wilfley DE. Assessment of binge eating in overweight youth using a questionnaire version of the child eating disorder examination with instructions. Int J Eat Disord. 2007;40(5):460–7.

    Article  PubMed  PubMed Central  Google Scholar 

  145. Parker K, Mitchell S, O’Brien P, Brennan L. Psychometric evaluation of disordered eating measures in bariatric surgery patients. Eat Behav. 2015;19:39–48.

    Article  PubMed  Google Scholar 

  146. Rø Ø, Reas DL, Rosenvinge J. The impact of age and BMI on Eating Disorder Examination Questionnaire (EDE-Q) scores in a community sample. Eat Behav. 2012;13(2):158–61.

    Article  PubMed  Google Scholar 

  147. Aardoom JJ, Dingemans AE, Op’t Landt MCS, Van Furth EF. Norms and discriminative validity of the Eating Disorder Examination Questionnaire (EDE-Q). Eat Behav. 2012;13(4):305–9.

    Article  PubMed  Google Scholar 

  148. Rø Ø, Reas DL, Stedal K. Eating disorder examination questionnaire (EDE-Q) in Norwegian adults: Discrimination between female controls and eating disorder patients. Eur Eat Disord Rev. 2015;23(5):408–12.

    Article  PubMed  Google Scholar 

  149. Gormally J, Black S, Daston S, Rardin D. The assessment of binge eating severity among obese persons. Addict Behav. 1982;7(1):47–55.

    Article  PubMed  Google Scholar 

  150. Fairburn CG, Cooper Z, O'Conner M. Eating Disorder Examination (EDE 17.0D) 2014.

  151. Sysko R, Glasofer DR, Hildebrandt T, Klimek P, Mitchell JE, Berg KC, et al. The Eating Disorder Assessment for DSM-5 (EDA-5): Development and validation of a structured interview for feeding and eating disorders. Int J Eat Disord. 2015;48(5):452–63.

    Article  PubMed  PubMed Central  Google Scholar 

  152. Van Strien T, Frijters JE, Bergers GP, Defares PB. The Dutch Eating Behavior Questionnaire (DEBQ) for assessment of restrained, emotional, and external eating behavior. Int J Eat Disord. 1986;5(2):295–315.

    Article  Google Scholar 

  153. Latner JD, Mond JM, Kelly MC, Haynes SN, Hay PJ. The loss of control over eating scale: development and psychometric evaluation. Int J Eat Disord. 2014;47(6):647–59.

    Article  PubMed  Google Scholar 

  154. Heriseanu AI, Hay P, Corbit L, Touyz S. Grazing in adults with obesity and eating disorders: a systematic review of associated clinical features and meta-analysis of prevalence. Clin Psychol Rev. 2017;58:16–32.

    Article  PubMed  Google Scholar 

  155. Heriseanu AI, Hay P, Touyz S. The short inventory of grazing (SIG): development and validation of a new brief measure of a common eating behaviour with a compulsive dimension. J Eat Disord. 2019;7(1):1–12.

    Article  Google Scholar 

  156. Lane B, Szabó M. Uncontrolled, repetitive eating of small amounts of food or ‘grazing’: development and evaluation of a new measure of atypical eating. Behav Change. 2013;30(2):57–73.

    Article  Google Scholar 

  157. Conceição EM, Mitchell JE, Machado PP, Vaz AR, Pinto-Bastos A, Ramalho S, et al. Repetitive eating questionnaire [Rep (eat)-Q]: enlightening the concept of grazing and psychometric properties in a Portuguese sample. Appetite. 2017;117:351–8.

    Article  PubMed  Google Scholar 

  158. Harris A, Hay P, Touyz S. Psychometric properties of instruments assessing exercise in patients with eating disorders: A systematic review. J Eat Disord. 2020;8(1):1–14.

    Article  Google Scholar 

  159. Pope HG Jr, Gruber AJ, Choi P, Olivardia R, Phillips KA. Muscle dysmorphia: an underrecognized form of body dysmorphic disorder. Psychosomatics. 1997;38(6):548–57.

    Article  PubMed  Google Scholar 

  160. Murray SB, Rieger E, Touyz SW, De la Garza GL, Yolanda. Muscle dysmorphia and the DSM-V conundrum: where does it belong? A review paper. Int J Eat Disord. 2010;43(6):483–91.

    Article  PubMed  Google Scholar 

  161. Cooper M, Eddy KT, Thomas JJ, Franko DL, Carron-Arthur B, Keshishian AC, et al. Muscle dysmorphia: a systematic and meta-analytic review of the literature to assess diagnostic validity. Int J Eat Disord. 2020;53(10):1583–604.

    Article  PubMed  Google Scholar 

  162. Murray SB, Griffiths S, Mond JM. Evolving eating disorder psychopathology: Conceptualising muscularity-oriented disordered eating. Br J Psychiatry. 2016;208(5):414–5.

    Article  PubMed  Google Scholar 

  163. Rohman L. The relationship between anabolic androgenic steroids and muscle dysmorphia: a review. Eat Disord. 2009;17(3):187–99.

    Article  PubMed  Google Scholar 

  164. García-Rodríguez J, Alvarez-Rayón G, Camacho-Ruíz J, Amaya-Hernández A, Mancilla-Díaz JM. Muscle dysmorphia and use of ergogenics substances. A systematic review. Rev Colomb Psiquiatr (English ed). 2017;46(3):168–77.

    Article  Google Scholar 

  165. Zickgraf HF, Ellis JM. Initial validation of the nine item avoidant/restrictive food intake disorder screen (NIAS): A measure of three restrictive eating patterns. Appetite. 2018;123:32–42.

    Article  PubMed  Google Scholar 

  166. Dovey TM, Aldridge VK, Martin CI, Wilken M, Meyer C. Screening Avoidant/Restrictive Food Intake Disorder (ARFID) in children: outcomes from utilitarian versus specialist psychometrics. Eat Behav. 2016;23:162–7.

    Article  PubMed  Google Scholar 

  167. Cecilia-Costa R, Volkening L, Laffel L. Factors associated with disordered eating behaviours in adolescents with Type 1 diabetes. Diabet Med. 2019;36(8):1020–7.

    Article  PubMed  PubMed Central  Google Scholar 

  168. Herpertz S, Albus C, Lichtblau K, Köhle K, Mann K, Senf W. Relationship of weight and eating disorders in type 2 diabetic patients: a multicenter study. Int J Eat Disord. 2000;28(1):68–77.

    Article  PubMed  Google Scholar 

  169. Wisting L, Wonderlich J, Skrivarhaug T, Dahl-Jørgensen K, Rø Ø. Psychometric properties and factor structure of the diabetes eating problem survey—revised (DEPS-R) among adult males and females with type 1 diabetes. J Eat Disord. 2019;7(1):2198–202.

    Article  Google Scholar 

  170. Zuijdwijk CS, Pardy SA, Dowden JJ, Dominic AM, Bridger T, Newhook LA. The mSCOFF for screening disordered eating in pediatric type 1 diabetes. Diabetes Care. 2014;37(2):26–7.

    Article  Google Scholar 

  171. Pursey KM, Hart M, Jenkins L, McEvoy M, Smart CE. Screening and identification of disordered eating in people with type 1 diabetes: a systematic review. J Diabetes Complicat. 2020;34(4): 107522.

    Article  Google Scholar 

  172. Project Implicit. Implicit Association Test: weight.

  173. Kinavey H, Cool C. The broken lens: how anti-fat bias in psychotherapy is harming our clients and what to do about it. Women Ther. 2019;42(1–2):116–30.

    Article  Google Scholar 

  174. Pearl RL, Puhl RM. Measuring internalized weight attitudes across body weight categories: validation of the modified weight bias internalization scale. Body Image. 2014;11(1):89–92.

    Article  PubMed  Google Scholar 

  175. Forney KJ, Brown TA, Holland-Carter LA, Kennedy GA, Keel PK. Defining “significant weight loss” in atypical anorexia nervosa. Int J Eat Disord. 2017;50(8):952–62.

    Article  PubMed  Google Scholar 

  176. Peebles R, Hardy KK, Wilson JL, Lock JD. Are diagnostic criteria for eating disorders markers of medical severity? Pediatrics. 2010;125(5):e1193–201.

    Article  PubMed  Google Scholar 

  177. World Health Organization. Framework for action on interprofessional education and collaborative practice. Geneva: World Health Organization; 2010.

    Google Scholar 

  178. Heruc G, Hurst K, Casey A, Fleming K, Freeman J, Fursland A, et al. ANZAED eating disorder treatment principles and general clinical practice and training standards. J Eat Disord. 2020;8(1):1–9.

    Google Scholar 

  179. Agras WS, Telch CF, Arnow B, Eldredge K, Detzer MJ, Henderson J, et al. Does interpersonal therapy help patients with binge-eating disorder who fail to respond to cognitive-behavioral therapy. J Consult Clin Psychol. 1995;63(3):356–60.

    Article  PubMed  Google Scholar 

  180. Eldredge KL, Agras WS, Arnow B, Telch CF, Bell S, Castonguay L, et al. The effects of extending cognitive-behavioral therapy for binge eating disorder among initial treatment nonresponders. Int J Eat Disord. 1997;21(4):347–52.

    Article  PubMed  Google Scholar 

  181. Gorin AA, Le Grange D, Stone AA. Effectiveness of spouse involvement in cognitive behavioral therapy for binge eating disorder. Int J Eat Disord. 2003;33(4):421–33.

  182. Lewer M, Kosfelder J, Michalak J, Schroeder D, Nasrawi N, Vocks S. Effects of a cognitive-behavioral exposure-based body image therapy for overweight females with binge eating disorder: a pilot study. J Eat Disord. 2017;5(1):1–12.

    Article  Google Scholar 

  183. Grilo CM, Masheb RM. A randomized controlled comparison of guided self-help cognitive behavioral therapy and behavioral weight loss for binge eating disorder. Behav Res Ther. 2005;43.

  184. Grilo CM, White MA, Gueorguieva R, Barnes RD, Masheb RM. Self-help for binge eating disorder in primary care: A randomized controlled trial with ethnically and racially diverse obese patients. 2013;51:855–61.

  185. Grilo CM, Masheb RM, White MA, Gueorguieva R, Barnes RD, Walsh BT, et al. Treatment of binge eating disorder in racially and ethnically diverse obese patients in primary care: Randomized placebo-controlled clinical trial of self-help and medication. Behav Res Ther. 2014;58:1–9.

  186. Shelley-Ummenhofer J, MacMillan PD. Cognitive-behavioural treatment for women who binge eat. Can J Diet Pract Res. 2007;68(3):139–42.

    Article  PubMed  Google Scholar 

  187. Alfonsson S, Parling T, Ghaderi A. Group behavioral activation for patients with severe obesity and binge eating disorder: a randomized controlled trial. Behav Modif. 2015;39(2):270–94.

    Article  PubMed  Google Scholar 

  188. Dastan B, Zanjani SA, Adl AF, Habibi M. The effectiveness of dialectical behaviour therapy for treating women with obesity suffering from BED: a feasibility and pilot study. Clin Psychol. 2020;24(2):133–42.

    Article  Google Scholar 

  189. Rahmani M, Omidi A, Asemi Z, Akbari H. The effect of dialectical behaviour therapy on binge eating, difficulties in emotion regulation and BMI in overweight patients with binge-eating disorder: a randomized controlled trial. Ment Health Prevent. 2018;9:13–8.

    Article  Google Scholar 

  190. Gaudiani JL, editor. And on towards the sea: Committment to weight-inclusive care imrpoves our patients' lives and our own. Australia and New Zealand Academy for Eating Disorders 2021 hybrid conference; 2021; Perth.

  191. Lammers MW, Vroling MS, Crosby RD, van Strien T. Dialectical behavior therapy adapted for binge eating compared to cognitive behavior therapy in obese adults with binge eating disorder: a controlled study. J Eat Disord. 2020;8:27.

    Article  PubMed  PubMed Central  Google Scholar 

  192. Wilfley DE, Welch RR, Stein RI, Spurrell EB, Cohen LR, Saelens BE, et al. A randomized comparison of group cognitive-behavioral therapy and group interpersonal psychotherapy for the treatment of overweight individuals with binge-eating disorder. Arch Gen Psychiatry. 2002;59(8):713–21.

    Article  PubMed  Google Scholar 

  193. Hilbert A, Bishop ME, Stein RI, Tanofsky-Kraff M, Swenson AK, Welch RR, et al. Long-term efficacy of psychological treatments for binge eating disorder. Br J Psychiatry. 2012;200(3):232–7.

    Article  PubMed  PubMed Central  Google Scholar 

  194. Palavras MA, Hay P, Filho CA, Claudino A. The Efficacy of Psychological Therapies in Reducing Weight and Binge Eating in People with Bulimia Nervosa and Binge Eating Disorder Who Are Overweight or Obese-A Critical Synthesis and Meta-Analyses. Nutrients. 2017;9(3):299.

  195. Agras WS, Telch CF, Arnow B, Eldredge K, Wilfley DE, Raeburn SD, et al. Weight-loss, cognitive-behavioral, and desipramine treatments in binge-eating disorder—an additive design. Behav Ther. 1994;25(2):225–38.

    Article  Google Scholar 

  196. Munsch S, Biedert E, Meyer A, Michael T, Schlup B, Tuch A, et al. A randomized comparison of cognitive behavioral therapy and behavioral weight loss treatment for overweight individuals with binge eating disorder. Int J Eat Disord. 2007;40(2):102–13.

    Article  PubMed  Google Scholar 

  197. Grilo CM, Masheb RM, Wilson GT, Gueorguieva R, White MA. Cognitive-behavioral therapy, behavioral weight loss, and sequential treatment for obese patients with binge-eating disorder: A randomized controlled trial. J Consult Clin Psychol. 2011;79(5):675–85.

  198. Nauta H, Hospers H, Kok G, Jansen A. A comparison between a cognitive and a behavioral treatment for obese binge eaters and obese non-binge eaters. Behav Ther. 2000;31(3):441–61.

    Article  Google Scholar 

  199. Munsch S, Meyer AH, Biedert E. Efficacy and predictors of long-term treatment success for cognitive-behavioral treatment and behavioral weight-loss-treatment in overweight individuals with binge eating disorder. Behav Res Ther. 2012;50(12):775–85.

    Article  PubMed  Google Scholar 

  200. Grilo CM, White MA, Masheb RM, Ivezaj V, Morgan PT, Gueorguieva R. Randomized controlled trial testing the effectiveness of adaptive “SMART” stepped-care treatment for adults with binge-eating disorder comorbid with obesity. Am Psychol. 2020;75(2):204–18.

    Article  PubMed  PubMed Central  Google Scholar 

  201. Palavras MA, Hay P, Mannan H, da Luz FQ, Sainsbury A, Touyz S, et al. Integrated weight loss and cognitive behavioural therapy (CBT) for the treatment of recurrent binge eating and high body mass index: a randomized controlled trial. Eat Weight Disord Stud Anorex Bulim Obes. 2021;26(1):249–62.

    Article  Google Scholar 

  202. Palavras MA, Hay P, Touyz S, Sainsbury A, da Luz F, Swinbourne J, et al. Comparing cognitive behavioural therapy for eating disorders integrated with behavioural weight loss therapy to cognitive behavioural therapy-enhanced alone in overweight or obese people with bulimia nervosa or binge eating disorder: study protocol for a randomised controlled trial. Trials. 2015;16(1):1–10.

    Article  Google Scholar 

  203. Cooper Z, Calugi S, Dalle GR. Controlling binge eating and weight: a treatment for binge eating disorder worth researching? Eat Weight Disord Stud Anorex Bulim Obes. 2020;25(4):1105–9.

    Article  Google Scholar 

  204. Mercado D, Robinson L, Gordon G, Werthmann J, Campbell IC, Schmidt U. The outcomes of mindfulness-based interventions for obesity and binge eating disorder: a meta-analysis of randomised controlled trials. Appetite. 2021;166: 105464.

    Article  Google Scholar 

  205. Brambilla F, Samek L, Company M, Lovo F, Cioni L, Mellado C. Multivariate therapeutic approach to binge-eating disorder: Combined nutritional, psychological and pharmacological treatment. Int Clin Psychopharmacol. 2009;24(6):312–7.

    Article  PubMed  Google Scholar 

  206. de Zwaan M, Mitchell JE, Crosby RD, Mussell MP, Raymond NC, Specker SM, et al. Short-term cognitive behavioral treatment does not improve outcome of a comprehensive very-low-calorie diet program in obese women with binge eating disorder. Behav Ther. 2005;36:89–99.

  207. Cesa GL, Manzoni GM, Bacchetta M, Castelnuovo G, Conti S, Gaggioli A, et al. Virtual reality for enhancing the cognitive behavioral treatment of obesity with binge eating disorder: randomized controlled study with one-year follow-up. 2013;15(6):139–51.

  208. Goodrick GK, Poston IWSC, Kimball KT, Reeves RS, Foreyt JP. Nondieting versus dieting treatment for overweight binge-eating women. J Consult Clin Psychol. 1998;66(2):363–8.

    Article  PubMed  Google Scholar 

  209. Golden NH, Katzman DK, Sawyer SM, Ornstein RM. Position paper of the society for adolescent health and medicine: medical management of restrictive eating disorders in adolescents and young adults. J Adolesc Health. 2015;56(1):121–5.

    Article  PubMed  Google Scholar 

  210. Seetharaman S, Golden NH, Halpern-Felsher B, Peebles R, Payne A, Carlson JL. Effect of a prior history of overweight on return of menses in adolescents with eating disorders. J Adolesc Health. 2017;60(4):469–71.

    Article  PubMed  Google Scholar 

  211. McElroy SL, Arnold LM, Shapira NA, Keck PE Jr, Rosenthal NR, Karim MR, et al. Topiramate in the treatment of binge eating disorder associated with obesity: A randomized, placebo-controlled trial. Am J Psychiatry. 2003;160(2):255–61.

    Article  PubMed  Google Scholar 

  212. Guerdjikova AI, McElroy SL, Welge JA, Nelson E, Keck PE, Hudson JI. Lamotrigine in the treatment of binge-eating disorder with obesity: a randomized, placebo-controlled monotherapy trial. Int Clin Psychopharmacol. 2009;24(3):150–8.

    Article  PubMed  Google Scholar 

  213. Barton BB, Segger F, Fischer K, Obermeier M, Musil R. Update on weight-gain caused by antipsychotics: a systematic review and meta-analysis. Expert Opin Drug Saf. 2020;19(3):295–314.

    Article  PubMed  Google Scholar 

  214. De Hert M, Yu W, Detraux J, Sweers K, van Winkel R, Correll CU. Body weight and metabolic adverse effects of asenapine, iloperidone, lurasidone and paliperidone in the treatment of schizophrenia and bipolar disorder. CNS Drugs. 2012;26(9):733–59.

    Article  PubMed  Google Scholar 

  215. McElroy SL, Guerdjikova A, Kotwal R, Welge JA, Nelson EB, Lake KA, et al. Atomoxetine in the treatment of binge-eating disorder: a randomized placebo-controlled trial. J Clin Psychiatry. 2007;68(3):390.

    Article  PubMed  Google Scholar 

  216. McElroy SL, Guerdjikova AI, Mori N, Blom TJ, Williams S, Casuto LS, et al. Armodafinil in binge eating disorder: a randomized, placebo-controlled trial. Int Clin Psychopharmacol. 2015;30(4):209–15.

    Article  PubMed  Google Scholar 

  217. McElroy SL, Hudson JI, Grilo CM, Guerdjikova AI, Deng L, Koblan KS, et al. Efficacy and safety of dasotraline in adults with binge-eating disorder: a randomized, placebo-controlled, flexible-dose clinical trial. J Clin Psychiatry. 2020;81(5):1–13.

    Article  Google Scholar 

  218. Grilo CM, McElroy SL, Hudson JI, Tsai J, Navia B, Goldman R, et al. Efficacy and safety of dasotraline in adults with binge-eating disorder: a randomized, placebo-controlled, fixed-dose clinical trial. CNS Spectr. 2020;26(5):481–90.

    Article  PubMed  PubMed Central  Google Scholar 

  219. Citrome L, Tsai J, Mandel M, Deng L, Grinnell T, Pikalov A. Effect of dasotraline on body weight in patients with binge-eating disorder. CNS Spectrums. 2019;25(2):307.

  220. Deb KS, Gupta R, Varshney M. Orlistat abuse in a case of bulimia nervosa: the changing Indian society. Gen Hosp Psychiatry. 2014;36(5):543–9.

    Article  Google Scholar 

  221. Tam G, Yeung MPS. A systematic review of the long-term effectiveness of work-based lifestyle interventions to tackle overweight and obesity. Prev Med. 2018;107:54–60.

    Article  PubMed  Google Scholar 

  222. Alcaraz-Ibáñez M, Paterna A, Sicilia Á, Griffiths MD. Morbid exercise behaviour and eating disorders: a meta-analysis. J Behav Addict. 2020;9(2):206–24.

    Article  PubMed  PubMed Central  Google Scholar 

  223. Gorrell S, Flatt RE, Bulik CM, Le Grange D. Psychosocial etiology of maladaptive exercise and its role in eating disorders: a systematic review. Int J Eat Disord. 2021;54(8):1358–76.

    Article  PubMed  PubMed Central  Google Scholar 

  224. Noetel M, Dawson L, Hay P, Touyz S. The assessment and treatment of unhealthy exercise in adolescents with anorexia nervosa: a Delphi study to synthesize clinical knowledge. Int J Eat Disord. 2017;50(4):378–88.

    Article  PubMed  Google Scholar 

  225. Levine MD, Marcus MD, Moulton P. Exercise in the treatment of binge eating disorder. Int J Eat Disord. 1996;19(2):171–7.

    Article  PubMed  Google Scholar 

  226. Pendleton VR, Goodrick GK, Poston WSC, Reeves RS, Foreyt JP. Exercise augments the effects of cognitive-behavioral therapy in the treatment of binge eating. Int J Eat Disord. 2002;31(2):172–84.

  227. McIver S, O’Halloran P, McGartland M. Yoga as a treatment for binge eating disorder: A preliminary study. Complement Ther Med. 2009;17(4):196–202.

    Article  PubMed  Google Scholar 

  228. Cook B, Wonderlich SA, Mitchell J, Thompson R, Sherman R, McCallum K. Exercise in eating disorders treatment: systematic review and proposal of guidelines. Med Sci Sports Exerc. 2016;48(7):1408–14.

    Article  PubMed  PubMed Central  Google Scholar 

  229. Shaw KA, Gennat HC, O'Rourke P, Mar CD. Exercise for overweight or obesity. Cochrane Database Syst Rev. 2006(4):Art. No.: CD003817.

  230. Elkington TJ, Cassar S, Nelson AR, Levinger I. Psychological responses to acute aerobic, resistance, or combined exercise in healthy and overweight individuals: a systematic review. Clin Med Insights Cardiol. 2017;11:1–23.

    Article  Google Scholar 

  231. Ruotsalainen H, Kyngäs H, Tammelin T, Kääriäinen M. Systematic review of physical activity and exercise interventions on body mass indices, subsequent physical activity and psychological symptoms in overweight and obese adolescents. J Adv Nurs. 2015;71(11):2461–77.

    Article  PubMed  Google Scholar 

  232. National Health Medical Research Council. Clinical practice guidelines for the management of overweight and obesity in adults, adolescents and children in Australia 2013 updated 2013.

  233. Kvam S, Kleppe CL, Nordhus IH, Hovland A. Exercise as a treatment for depression: a meta-analysis. J Affect Disord. 2016;202:67–86.

    Article  PubMed  Google Scholar 

  234. Hughes EK, Le Grange D, Court A, Sawyer SM. A case series of family-based treatment for adolescents with atypical anorexia nervosa. Int J Eat Disord. 2017;50(4):424–32.

    Article  PubMed  Google Scholar 

  235. Dimitropoulos G, Kimber M, Singh M, Williams EP, Loeb KL, Hughes EK, et al. Stay the course: Practitioner reflections on implementing family-based treatment with adolescents with atypical anorexia. J Eat Disord. 2019;7(1):1–11.

    Article  Google Scholar 

  236. Lie SØ, Rø Ø, Bang L. Is bullying and teasing associated with eating disorders? A systematic review and meta-analysis. Int J Eat Disord. 2019;52(5):497–514.

    Article  PubMed  Google Scholar 

  237. Dahill LM, Touyz S, Morrison NM, Hay P. Parental appearance teasing in adolescence and associations with eating problems: a systematic review. BioMed Central Public Health. 2021;21(1):1–13.

    Article  Google Scholar 

  238. Shomaker LB, Tanofsky-Kraff M, Matherne CE, Mehari RD, Olsen CH, Marwitz SE, et al. A randomized, comparative pilot trial of family-based interpersonal psychotherapy for reducing psychosocial symptoms, disordered-eating, and excess weight gain in at-risk preadolescents with loss-of-control-eating. Int J Eat Disord. 2017;50(9):1084–94.

    Article  PubMed  PubMed Central  Google Scholar 

  239. Jeffrey S, Heruc G. Balancing nutrition management and the role of dietitians in eating disorder treatment. J Eat Disord. 2020;8(1):1–3.

    Article  Google Scholar 

  240. Independent Hospital Pricing Authority. International Statistical Classification of Diseases and Related Health Problems, Tenth Revision, Australian Modification (ICD-10-AM) 2019.

  241. American Academy of Pediatrics. Statement of endorsement: Defining pediatric malnutrition. Pediatrics. 2013;132(1):283.

  242. Mehta NM, Corkins MR, Lyman B, Malone A, Goday PS, Carney L, et al. Defining pediatric malnutrition. J Parenter Enter Nutr. 2013;37(4):460–81.

    Article  Google Scholar 

  243. Hickman IT, Tapsell L. Evidence based practice guidelines for the nutritional management of malnutrition in adult patients across the continuum of care. Nutr Diet. 2009;66:1–34.

    Article  Google Scholar 

  244. Kaidar-Person O, Person B, Szomstein S, Rosenthal RJ. Nutritional deficiencies in morbidly obese patients: a new form of malnutrition? Obes Surg. 2008;18(7):870–6.

    Article  PubMed  Google Scholar 

  245. García OP, Long KZ, Rosado JL. Impact of micronutrient deficiencies on obesity. Nutr Rev. 2009;67(10):559–72.

    Article  PubMed  Google Scholar 

  246. Kaidar-Person O, Person B, Szomstein S, Rosenthal RJ. Nutritional deficiencies in morbidly obese patients: a new form of malnutrition? Obes Surg. 2008;18(8):1028–34.

    Article  PubMed  Google Scholar 

  247. Forouhi NG, Misra A, Mohan V, Taylor R, Yancy W. Dietary and nutritional approaches for prevention and management of type 2 diabetes. Br Med J. 2018;361:1–9.

    Google Scholar 

  248. Cohen JB. Hypertension in obesity and the impact of weight loss. Curr Cardiol Rep. 2017;19(10):1–8.

    Article  Google Scholar 

  249. Klop B, Elte JWF, Cabezas MC. Dyslipidemia in obesity: mechanisms and potential targets. Nutrients. 2013;5(4):1218–40.

    Article  PubMed  PubMed Central  Google Scholar 

  250. Ochner CN, Barrios DM, Lee CD, Pi-Sunyer FX. Biological mechanisms that promote weight regain following weight loss in obese humans. Physiol Behav. 2013;120:106–13.

    Article  PubMed  Google Scholar 

  251. Bacon L, Aphramor L. Weight science: evaluating the evidence for a paradigm shift. Nutr J. 2011;10(1):1–13.

    Google Scholar 

  252. Bozzetto L, Costabile G, Della Pepa G, Ciciola P, Vetrani C, Vitale M, et al. Dietary fibre as a unifying remedy for the whole spectrum of obesity-associated cardiovascular risk. Nutrients. 2018;10(7):943–76.

    Article  PubMed Central  Google Scholar 

  253. Grillo A, Salvi L, Coruzzi P, Salvi P, Parati G. Sodium intake and hypertension. Nutrients. 2019;11(9):1970.

    Article  PubMed Central  Google Scholar 

  254. Peckmezian T, Hay P. A systematic review and narrative synthesis of interventions for uncomplicated obesity: weight loss, well-being and impact on eating disorders. J Eat Disord. 2017;5(1):1–15.

    Article  Google Scholar 

  255. Moustafa AF, Quigley KM, Wadden TA, Berkowitz RI, Chao AM. A systematic review of binge eating, loss of control eating, and weight loss in children and adolescents. Obesity. 2021;29(8):1259–71.

    Article  PubMed  Google Scholar 

  256. Gow ML, Tee MS, Garnett SP, Baur LA, Aldwell K, Thomas S, et al. Pediatric obesity treatment, self-esteem, and body image: a systematic review with meta-analysis. Pediatr Obes. 2020;15(3): e12600.

    Article  PubMed  Google Scholar 

  257. Da Luz F, Hay P, Gibson AA, Touyz SW, Swinbourne JM, Roekenes JA, et al. Does severe dietary energy restriction increase binge eating in overweight or obese individuals? A systematic review. Obes Rev. 2015;16(8):652–65.

    Article  PubMed  Google Scholar 

  258. Goebel-Fabbri AE. Prevention and recovery from eating disorders in type 1 diabetes: injecting. Hope: Taylor & Francis; 2017.

    Book  Google Scholar 

  259. Statewide Diabetes Clinical Network. Disordered Eating (DE) and Eating Disorders (ED) in Adults with Type 1 Diabetes (T1D) (aged 16 years and over). Document No. D5.01-V1-P21-R24. 2021.

  260. Lee I, Cooney LG, Saini S, Smith ME, Sammel MD, Allison KC, et al. Increased risk of disordered eating in polycystic ovary syndrome. Fertil Steril. 2017;107(3):796–802.

    Article  PubMed  Google Scholar 

  261. Jeanes YM, Reeves S, Gibson EL, Piggott C, May VA, Hart KH. Binge eating behaviours and food cravings in women with polycystic ovary syndrome. Appetite. 2017;109:24–32.

    Article  PubMed  Google Scholar 

  262. Krug I, Giles S, Paganini C. Binge eating in patients with polycystic ovary syndrome: prevalence, causes, and management strategies. Neuropsychiatr Dis Treat. 2019;15:1273–85.

    Article  PubMed  PubMed Central  Google Scholar 

  263. Fassio A, Idolazzi L, Rossini M, Gatti D, Adami G, Giollo A, et al. The obesity paradox and osteoporosis. Eat Weight Disord Stud Anorex Bulim Obes. 2018;23(3):293–302.

    Article  Google Scholar 

  264. Nagata JM, Carlson JL, Golden NH, Long J, Murray SB, Peebles R. Comparisons of bone density and body composition among adolescents with anorexia nervosa and atypical anorexia nervosa. Int J Eat Disord. 2019;52(5):591–6.

    Article  PubMed  PubMed Central  Google Scholar 

  265. Bachmann KN, Schorr M, Bruno AG, Bredella MA, Lawson EA, Gill CM, et al. Vertebral volumetric bone density and strength are impaired in women with low-weight and atypical anorexia nervosa. J Clin Endocrinol Metab. 2016;102(1):57–68.

    PubMed Central  Google Scholar 

  266. Schvey NA, Tanofsky-Kraff M, Yanoff LB, Checchi JM, Shomaker LB, Brady S, et al. Disordered-eating attitudes in relation to bone mineral density and markers of bone turnover in overweight adolescents. J Adolesc Health. 2009;45(1):33–9.

    Article  PubMed  PubMed Central  Google Scholar 

  267. Piya MK, Chimoriya R, Yu W, Grudzinskas K, Myint KP, Skelsey K, et al. Improvement in eating disorder risk and psychological health in people with class 3 obesity: effects of a multidisciplinary weight management program. Nutrients. 2021;13(5):1425–37.

    Article  PubMed  PubMed Central  Google Scholar 

  268. D’Souza C, Hay P, Touyz S, Piya MK. Bariatric and cosmetic surgery in people with eating disorders. Nutrients. 2020;12(9):2861.

    Article  PubMed Central  Google Scholar 

  269. Parker K, O’Brien P, Brennan L. Measurement of disordered eating following bariatric surgery: a systematic review of the literature. Obes Surg. 2014;24(6):945–53.

    Article  PubMed  Google Scholar 

  270. Parker K, Brennan L. Measurement of disordered eating in bariatric surgery candidates: a systematic review of the literature. Obes Res Clin Pract. 2015;9(1):12–25.

    Article  PubMed  Google Scholar 

  271. Parretti H, Hughes C, Jones L. ‘The rollercoaster of follow-up care’after bariatric surgery: a rapid review and qualitative synthesis. Obes Rev. 2019;20(1):88–107.

    Article  PubMed  Google Scholar 

  272. Segura-Garcia C, Caroleo M, Rania M, Barbuto E, Sinopoli F, Aloi M, et al. Binge eating disorder and bipolar spectrum disorders in obesity: psychopathological and eating behaviors differences according to comorbidities. J Affect Disord. 2017;208:424–30.

    Article  PubMed  Google Scholar 

  273. Kouidrat Y, Amad A, Lalau J-D, Loas G. Eating disorders in schizophrenia: implications for research and management. Schizophr Res Treat. 2014;2014:1–7.

    Article  Google Scholar 

  274. Alvarez-Jimenez M, Gonzalez-Blanch C, Crespo-Facorro B, Hetrick S, Rodriguez-Sanchez JM, Perez-Iglesias R, et al. Antipsychotic-induced weight gain in chronic and first-episode psychotic disorders. CNS Drugs. 2008;22(7):547–62.

    Article  PubMed  Google Scholar 

  275. Citrome L, Holt RI, Walker DJ, Hoffmann VP. Weight gain and changes in metabolic variables following olanzapine treatment in schizophrenia and bipolar disorder. Clin Drug Investig. 2011;31(7):455–82.

    Article  PubMed  Google Scholar 

  276. Fagiolini A, Chengappa KR. Weight gain and metabolic issues of medicines used for bipolar disorder. Curr Psychiatry Rep. 2007;9(6):521–8.

    Article  PubMed  Google Scholar 

  277. Treuer T, Hoffmann VP, Chen AK-P, Irimia V, Ocampo M, Wang G, et al. Factors associated with weight gain during olanzapine treatment in patients with schizophrenia or bipolar disorder: Results from a six-month prospective, multinational, observational study. World J Biol Psychiatry. 2009;10(4–3):729–40.

    Article  PubMed  Google Scholar 

  278. Blouin M, Tremblay A, Jalbert M-E, Venables H, Bouchard R-H, Roy M-A, et al. Adiposity and eating behaviors in patients under second generation antipsychotics. Obesity. 2008;16(8):1780–7.

    Article  PubMed  Google Scholar 

  279. Acle A, Cook BJ, Siegfried N, Beasley T. Cultural considerations in the treatment of eating disorders among racial/ethnic minorities: a systematic review. J Cross Cult Psychol. 2021.

    Article  Google Scholar 

  280. Murray SB, Nagata JM, Griffiths S, Calzo JP, Brown TA, Mitchison D, et al. The enigma of male eating disorders: a critical review and synthesis. Clin Psychol Rev. 2017;57:1–11.

    Article  PubMed  Google Scholar 

  281. Mitchison D, Mond J. Epidemiology of eating disorders, eating disordered behaviour, and body image disturbance in males: a narrative review. J Eat Disord. 2015;3(1):1–9.

    Article  Google Scholar 

  282. Hay P, Girosi F, Mond J. Prevalence and sociodemographic correlates of DSM-5 eating disorders in the Australian population. J Eat Disord. 2015;3(1):1–7.

    Article  Google Scholar 

  283. Eddy KT, Thomas JJ, Hastings E, Edkins K, Lamont E, Nevins CM, et al. Prevalence of DSM-5 avoidant/restrictive food intake disorder in a pediatric gastroenterology healthcare network. Int J Eat Disord. 2015;48(5):464–70.

    Article  PubMed  Google Scholar 

  284. Gueguen J, Godart N, Chambry J, Brun-Eberentz A, Foulon C, Divac P, Snezana M, et al. Severe anorexia nervosa in men: comparison with severe AN in women and analysis of mortality. Int J Eat Disord. 2012;45(4):537–45.

    Article  PubMed  Google Scholar 

  285. Griffiths S, Mond JM, Murray SB, Touyz S. Young peoples’ stigmatizing attitudes and beliefs about anorexia nervosa and muscle dysmorphia. Int J Eat Disord. 2014;47(2):189–95.

    Article  PubMed  Google Scholar 

  286. Striegel-Moore RH, Garvin V, Dohm FA, Rosenheck RA. Eating disorders in a national sample of hospitalized female and male veterans: detection rates and psychiatric comorbidity. Int J Eat Disord. 1999;25(4):405–14.

    Article  PubMed  Google Scholar 

  287. Currin L, Schmidt U, Waller G. Variables that influence diagnosis and treatment of the eating disorders within primary care settings: a vignette study. Int J Eat Disord. 2007;40(3):257–62.

    Article  PubMed  Google Scholar 

  288. Leit RA, Pope HG Jr, Gray JJ. Cultural expectations of muscularity in men: the evolution of Playgirl centerfolds. Int J Eat Disord. 2001;29(1):90–3.

    Article  PubMed  Google Scholar 

  289. Frederick DA, Buchanan GM, Sadehgi-Azar L, Peplau LA, Haselton MG, Berezovskaya A, et al. Desiring the muscular ideal: men’s body satisfaction in the United States, Ukraine, and Ghana. Psychol Men Masc. 2007;8(2):103.

    Article  Google Scholar 

  290. Hesse-Biber S, Leavy P, Quinn CE, Zoino J. The mass marketing of disordered eating and eating disorders: The social psychology of women, thinness and culture. Womens Stud Int Forum. 2006;29(2):208–24.

    Article  Google Scholar 

  291. Murray SB, Griffiths S, Rieger E, Touyz S. A comparison of compulsive exercise in male and female presentations of anorexia nervosa: what is the difference? Adv Eat Disord Theory Res Pract. 2014;2(1):65–70.

    Google Scholar 

  292. Thapliyal P, Hay P, Conti J. Role of gender in the treatment experiences of people with an eating disorder: a metasynthesis. J Eat Disord. 2018;6(1):1–16.

    Article  Google Scholar 

  293. Bunnell DW. Psychotherapy with men with eating disorders: The influence of gender socialization and masculine gender norms on engagement and treatment. In: Nagata JM, Brown TA, Murray SB, Lavender JM, editors. Eating Disorders in Boys and Men: Springer, Cham; 2021. p. 197–213.

  294. Lin I, Green C, Bessarab D. ‘Yarn with me’: applying clinical yarning to improve clinician–patient communication in Aboriginal health care. Aust J Prim Health. 2016;22(5):377–82.

    Article  PubMed  Google Scholar 

  295. Gee G, Dudgeon P, Schultz C, Hart A, Kelly K. Aboriginal and Torres Strait Islander social and emotional wellbeing. In: Dudgeon P, Milroy H, Walker R, editors. Working Together: Aboriginal and Torres Strait Islander Mental Health and Wellbeing Principles and Practice. 2. Canberra, Australia: Commonwealth Australia (collaboration with the University of Western Australia; 2014. p. 55–68.

  296. Temple JB, Russell J. Food insecurity among older Aboriginal and Torres Strait Islanders. Int J Environ Res Public Health. 2018;15(8):1766.

    Article  PubMed Central  Google Scholar 

  297. Walker R, Schultz C, Sonn C. Cultural competence–transforming policy, services, programs and practice. Barton, ACT: Australian Government Department of the Prime Minister and Cabinet; 2014. p. 195–220.

  298. Nagata JM, Ganson KT, Austin SB. Emerging trends in eating disorders among sexual and gender minorities. Curr Opin Psychiatry. 2020;33(6):562–7.

    Article  PubMed  PubMed Central  Google Scholar 

  299. Parker LL, Harriger JA. Eating disorders and disordered eating behaviors in the LGBT population: a review of the literature. J Eat Disord. 2020;8(1):1–20.

    Article  Google Scholar 

  300. Cuzzolaro M, Milano W. Gender identity, sexual orientation, body image, eating, and weight. In: Cuzzolaro M, Fassino S, editors. Body Image, Eating, and Weight: Springer, Cham; 2018. p. 261–72.

  301. Kozee HB, Tylka TL, Bauerband LA. Measuring transgender individuals’ comfort with gender identity and appearance: Development and validation of the Transgender Congruence Scale. Psychol Women Q. 2012;36(2):179–96.

    Article  Google Scholar 

  302. Craig SL, Austin A, Levenson J, Leung VW, Eaton AD, D’Souza SA. Frequencies and patterns of adverse childhood events in LGBTQ+ youth. Child Abuse Negl. 2020;107:1–12.

    Article  Google Scholar 

  303. Pantalone DW, Gorman KR, Pereida ET, Valentine SE. Trauma among sexual and gender minority populations. In: Rothblum ED, editor. The Oxford Handbook of Sexual and Gender Minority Mental Health. New York: Oxford University Press; 2020. p. 59–71.

  304. Mensinger JL, Granche JL, Cox SA, Henretty JR. Sexual and gender minority individuals report higher rates of abuse and more severe eating disorder symptoms than cisgender heterosexual individuals at admission to eating disorder treatment. Int J Eat Disord. 2020;53(4):541–54.

    Article  PubMed  PubMed Central  Google Scholar 

  305. Frost DM, Lehavot K, Meyer IH. Minority stress and the health of sexual minorities individuals. J Behav Med. 2015;38(1):1–8.

    Article  PubMed  Google Scholar 

  306. Pachankis JE, Clark KA, Burton CL, Hughto JMW, Bränström R, Keene DE. Sex, status, competition, and exclusion: Intraminority stress from within the gay community and gay and bisexual men’s mental health. J Pers Soc Psychol. 2020;119(3):713.

    Article  PubMed  PubMed Central  Google Scholar 

  307. Panza E, Olson K, Goldstein CM, Selby EA, Lillis J. Characterizing lifetime and daily experiences of weight stigma among sexual minority women with overweight and obesity: a descriptive study. Int J Environ Res Public Health. 2020;17(13):4892.

    Article  PubMed Central  Google Scholar 

  308. Austen E, Greenaway KH, Griffiths S. Differences in weight stigma between gay, bisexual, and heterosexual men. Body Image. 2020;35:30–40.

    Article  PubMed  Google Scholar 

  309. Duffy ME, Henkel KE, Earnshaw VA. Transgender clients’ experiences of eating disorder treatment. J LGBT Issues Couns. 2016;10(3):136–49.

    Article  PubMed  PubMed Central  Google Scholar 

  310. Zelle A, Arms T. Psychosocial effects of health disparities of lesbian, gay, bisexual, and transgender older adults. J Psychosoc Nurs Ment Health Serv. 2015;53(7):25–30.

    Article  PubMed  Google Scholar 

  311. Fairburn CG. Cognitive behavior therapy and eating disorders. New York: Guilford Press; 2008.

  312. Schmidt U, Startup H, Treasure J. A cognitive-interpersonal therapy workbook for treating anorexia nervosa: the Maudsley model. London: Routledge; 2018.

  313. McIntosh VV, Jordan J, Luty SE, Carter FA, McKenzie JM, Bulik CM, et al. Specialist supportive clinical management for anorexia nervosa. Int J Eat Disord. 2006;39(8):625–32.

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We would like to acknowledge the following individuals for their contributors to this guideline: Phillip Aouad, Katrina Baikie, Zoe Bower, Megan Bray, Emma Bryant, Liz Dale, Rosiel Elwyn, Annemarie Hindle, Jordan Martenstyn, Kate Mellody, Anita Raspovic, and Emily Unity. We also acknowledge the following organisations for their external reviews of Version 2 of this guideline: Australian & New Zealand Academy for Eating Disorders (ANZAED), Australian & New Zealand Academy for Eating Disorders Weight Stigma and Social Justice Special Interest Group (ANZAED WS&SJ SIG), Australian & New Zealand Metabolic and Obesity Surgery Society (ANZMOSS), Australian Psychological Society (APS), Centre of Excellence in Eating Disorders (CEED), Dietitians Australia, Healthy at Every Size Australia® (HAES®), National Association of Clinical Obesity Services (NACOS), The Obesity Collective, and The Royal Australian College of General Practitioners (RACGP). We would also like to thank the following external expert reviewers of Version 2: Catherine Bacus, Jennifer Conway, Susan Hart, Nic Kormas, Richard Newton, Elizabeth Rieger, Andrea Ryan, Amanda Salis, Fiona Willer, and Gary Wittert.


This guideline received funding from the Australian Government Department of Health.

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Authors and Affiliations



PH and members of the NEDC Steering Committee conceived the idea for this project. All authors were involved in drafting the manuscript. PH chaired the Guideline Development Group and PH and AFR were primarily responsible for the over project design, oversight of project and editing the manuscript following consultation and reviews. All authors contributed to finalising the recommendations. All authors read and edited the manuscript, and approved the final manuscript.

Corresponding author

Correspondence to Angelique F. Ralph.

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Competing interests

Leah Brennan: La Trobe University—Employee; Nourish Psychology (Centre for Eating, Weight and Body Image)—Director; NEDC—Steering Committee member; Australian Psychological Society—Member; Australian and New Zealand Academy of Eating Disorders—Member; SAX Institute—Past tender recipient; NHMRC, Diabetes Australia— Past grant recipient. Susan Byrne: NEDC—Steering Committee member; Australian Medical Council—Contracted employee; Takeda Pharmaceuticals—Sponsored speaker 2018; NHMRC, Healthway, Rotary—Past grant recipient; Butterfly Foundation—Clinical Advisory Committee. Belinda Caldwell: Eating Disorders Victoria—CEO; Eating Disorders Families Australia—Past board director/deputy chair; Families Empowered and Supporting Treatment of Eating Disorders (F.E.A.S.T)—Past executive director. Jo Farmer: Justice Connect—Contracted employee (until 9 April 2021); Beyond Blue—Consultant (until March 2021); Department of Social Services—Consultant (until June 2021); Mental Health Coordinating Council (NSW)—Consultant (until July 2021); National Mental Health Commission—Member, National Workplace Initiative Evaluation Working Group (ongoing); Queensland Mental Health Commission—Consultant (until December 2021); Zero Suicide Institute of Australasia—Faculty member; deliver training (ongoing); Australian Evaluation Society—Member; Blog editor. Laura Hart: University of Melbourne—Employee; La Trobe University—Honorary appointment; STRIPED Harvard/Boston Children’s Hospital—Honorary appointment; NEDC—Steering Committee member; Victorian Eating Disorders Research Network—Founder, Convenor; Body Confident Collective—Founder, Director; Australia New Zealand Academy for Eating Disorders—Member; Academy for Eating Disorders—Member; Society for Mental Health Research—Member; Comonground—Member; NHMRC, Australian Rotary Health—Current and past grant recipient. Phillipa Hay: Western Sydney University—Employee; Wesley Hospital Ashfield—Contracted Director Eating Disorder service; South Western Sydney Local Health District—Employee; NEDC—Chair Steering Committee; Australian Medical Council—Contracted employee; HETI/NSW Institute of Psychiatry—Contracted employee; NHMRC—Member Assigners Academy (occasional); Royal Australian and New Zealand College of Psychiatrists—Fellow; Academy for Eating Disorders—Fellow; Australian Medical Association—Member; Takeda Pharmaceuticals—2017 Contracted report, sponsored speaker 2018; BMC publishers—Editor in Chief (honoraria) and Assoc Editor; World Health Organization—Member ICD 11 Eating Disorders Working Group; Department of Health—Member MBS Review—Eating Disorders Working Group 2017; NHMRC, Australian Research Council, CAPES Foundation—Past grant recipient; Australian Medical Council—Contracted employee. Gabriella Heruc: Northern Sydney Local Health District—Employee; Appetite for Change—Director and head clinician of private practice; Australia and New Zealand Academy for Eating Disorders—Past-President and Executive Committee Member; Western Sydney University—Adjunct Fellow; NEDC—Steering Committee Member; Dietitians Association of Australia—Member; Department of Health—Member MBS Implementation Committee—Eating Disorder Management Plans 2019. Sarah Maguire: University of Sydney—Employee; Sydney Local Health District—Employee; NHMRC—External Reviewer; Academy for Eating Disorders—Member; NSW Health—Advisor; NEDC—Steering Committee member; BMC publishers—Editor; Department of Health—Member MBS Review—Eating Disorders Working Group 2017, Funded project lead. Milan Piya: Western Sydney University—Employee; South Western Sydney Local Health District—Employee; Royal Australasian College of Physicians—Fellow; Australian Diabetes Society—Paid Member; Australian and New Zealand Obesity Society—Paid Member; Diabetes UK—Paid Member; Journal of Eating Disorders (BMC Publishers)—Senior Editor; Nutrients Journal (MDPI Publishers)—Guest Editor for special issue 2020–2022; Diabetes, Nutrition and Dietetics (Frontier Journals)—Associate Editor; Takeda Pharmaceuticals—Sponsored speaker 2019 and 2021; Novo Nordisk—Sponsored speaker 2020, 2021 and 2022; Johnson and Johnson Medical Pty Ltd—Advisory focus group 2021; Inova Pharmaceuticals—Sponsored online conference 2021; Eli Lilly and Company—Sponsored Speaker 2021; UCB Pharmaceuticals—Sponsored speaker 2020. Julia Quin: Eating Disorders Victoria—Carer Coach; EDV Rally Parent Support Group—co-facilitator; International Eating Disorders Family Support (IEDFS)—IEDFS—Facebook Group Administrator. Angelique Ralph: NEDC—Employee; Mind Plasticity—Sub-contractor; La Trobe University—Adjunct Research Fellow; Australian Clinical Psychology Association—Member; ANZAED—Member; NHMRC—Past grant recipient. Sarah Trobe: NEDC—Employee; The Victorian Centre of Excellence in Eating Disorders—Past employee; Royal Children’s Hospital—Past employee. Andrew Wallis: Sydney Children's Hospital Network—Employee; NEDC—Steering Committee member; ADW Training and Supervision—Director; Australian Association of Social Workers—Member. AJ Williams-Tchen: MHFA—Mental Health First Aid Australia—Current MHFA Instructor, Former employee; Butterfly Foundation—Reconciliation Action Plan Member; Reference Group, Ambassador; Beyond Blue—Reconciliation Action Plan Member; Consultant; ANZAED—Cultural Consultant; Royal Australian College of General Practitioners—Continuing Professional Development (CPD) Provider; College of Midwives—CPD Provider; Australian College of Rural and Remote Medicine—CPD Provider; Australian Association of Social Workers—Mental Health Accreditation; CPD Provider; South Eastern Melbourne Primary Health Network (PHN), North Western Melbourne PHN, Eastern Melbourne PHN, Gippsland PHN—Funding recipient 2020; Girraway Ganyi Consultancy—Director; Fellowship Victorian Indigenous leadership—Emerging Leader 2015–2016; Victorian Aboriginal Community Health Organisations across Victoria—Contractor for MHFA training; Cancer Council Victoria—Cultural Mentor since 2012, Independent Schools Victoria— CPD Provider; Cultural Consultant.

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Additional file 1.

Appendices. Appendix A - D.

Additional file 2.

RIGHT Checklist. RIGHT (Reporting Items for Practice Guidelines in Healthcare) Statement for Practice Guidelines checklist.

Additional file 3.

Quality appraisal. Quality appraisal of systematic reviews and meta-analyses appraised using JBI critical appraisal checklist for systematic reviews and research syntheses.

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